Expression pattern of candidate cell death effector proteins Bax, Bcl-2, Bcl-X, and c-Jun in sensory and motor neurons following sciatic nerve transection in the rat. (Q48862669)

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English	Expression pattern of candidate cell death effector proteins Bax, Bcl-2, Bcl-X, and c-Jun in sensory and motor neurons following sciatic nerve transection in the rat.	scientific article

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scholarly article

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Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8955944

Expression pattern of candidate cell death effector proteins Bax, Bcl-2, Bcl-X, and c-Jun in sensory and motor neurons following sciatic nerve transection in the rat. (English)

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8955944

1 reference

based on heuristic

inferred from title

Lars Klimaschewski

2

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8955944

author name string

Gillardon F

1

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8955944

Wickert H

3

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8955944

Krajewski S

4

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8955944

Reed JC

5

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8955944

Zimmermann M

6

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Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8955944

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1 November 1996

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Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8955944

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Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8955944

739

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Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8955944

1-2

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16 February 2018

http://europepmc.org/abstract/MED/8955944

244-250

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Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8955944

Nerve cell degeneration and death in the trigeminal ganglion of the adult rat following peripheral nerve transection.

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Role of Bcl-2 in the brain-derived neurotrophic factor survival response

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bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell death

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Temporal analysis of events associated with programmed cell death (apoptosis) of sympathetic neurons deprived of nerve growth factor

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The differential control of c-jun expression in regenerating sensory neurons and their associated glial cells.

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The role of inducible transcription factors in apoptotic nerve cell death

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Expression of mRNAs for neurotrophin receptors in the dorsal root ganglion and spinal cord during development and following peripheral or central axotomy.

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Altered gene expression in neurons during programmed cell death: identification of c-jun as necessary for neuronal apoptosis

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bcl-2 transgene expression can protect neurons against developmental and induced cell death

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Differential regulation of bcl-2, bax, c-fos, junB, and krox-24 expression in the cerebellum of Purkinje cell degeneration mutant mice

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Differential expression of bcl-2 and bax mRNA in axotomized dorsal root ganglia of young and adult rats

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Role of BCL-2 in the survival and function of developing and mature sympathetic neurons

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A c-Jun dominant negative mutant protects sympathetic neurons against programmed cell death

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The transcription factors c-JUN, JUN D and CREB, but not FOS and KROX-24, are differentially regulated in axotomized neurons following transection of rat sciatic nerve

1 reference

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Death of some dorsal root ganglion neurons and plasticity of others following sciatic nerve section in adult and neonatal rats

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Expression of different Jun and Fos proteins during the G0-to-G1 transition in mouse fibroblasts: in vitro and in vivo associations

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Upregulation of bax protein levels in neurons following cerebral ischemia.

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Selective expression of Jun proteins following axotomy and axonal transport block in peripheral nerves in the rat: evidence for a role in the regeneration process

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7 January 2021

Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

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The influence of regeneration and nerve growth factor on the neuronal cell body reaction to injury

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Expression of mRNA for neurotrophic factors and their receptors in the rat dorsal root ganglion and sciatic nerve following nerve injury

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7 January 2021

Multiple Bcl-2 family members demonstrate selective dimerizations with Bax

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Cell death: the significance of apoptosis

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BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with Bax.

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7 January 2021

bcl-2 inhibits death of central neural cells induced by multiple agents

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7 January 2021

Neurones and glial cells of the mouse sciatic nerve undergo apoptosis after injury in vivo and in vitro

1 reference

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7 January 2021

Structure-function analysis of Bcl-2 protein. Identification of conserved domains important for homodimerization with Bcl-2 and heterodimerization with Bax.

1 reference

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7 January 2021

The biochemistry of cell death by apoptosis

1 reference

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7 January 2021

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Identifiers

10.1016/S0006-8993(96)00829-3

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8955944

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8955944

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