Ammonium acetate challenge in experimental chronic hepatic encephalopathy induces a transient increase of brain 5-HT release in vivo. (Q48869475)

16 February 2018

title

Ammonium acetate challenge in experimental chronic hepatic encephalopathy induces a transient increase of brain 5-HT release in vivo. (English)

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16 February 2018

main subject

hepatic encephalopathy

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6

Butterworth RF

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16 February 2018

author name string

Bergqvist PB

1

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16 February 2018

Hjorth S

2

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16 February 2018

Audet RM

3

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16 February 2018

Apelqvist G

4

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16 February 2018

Bengtsson F

5

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16 February 2018

language of work or name

English

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publication date

1 November 1996

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16 February 2018

European Neuropsychopharmacology

published in

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16 February 2018

volume

6

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16 February 2018

issue

4

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16 February 2018

page(s)

317-322

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16 February 2018

Brain monoamine metabolism and behavior in portacaval-shunted rats.

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Brain tryptophan hydroxylation in the portacaval shunted rat: a hypothesis for the regulation of serotonin turnover in vivo

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Monoamines and metabolites in autopsied brain tissue from cirrhotic patients with hepatic encephalopathy

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Effect of Ammonia on Brain Serotonin Metabolism in Relation to Function in the Portacaval Shunted Rat

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Neocortical dialysate monoamines of rats after acute, subacute, and chronic liver shunt

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Acute effects of L-tryptophan on brain extracellular 5-HT and 5-HIAA levels in chronic experimental portal-systemic encephalopathy

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The effect of ammonia infusion on brain monoamine metabolism in portacaval-shunted rats

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Ammonia-induced release of neurotransmitters from rat brain synaptosomes: differences between the effects on amines and amino acids.

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Amino acid changes in regions of the CNS in relation to function in experimental portal-systemic encephalopathy

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Serotonin 5-HT1A autoreceptor blockade potentiates the ability of the 5-HT reuptake inhibitor citalopram to increase nerve terminal output of 5-HT in vivo: a microdialysis study.

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Brain monoamines after portacaval anastomosis.

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Amphetamine and other psychostimulants reduce pH gradients in midbrain dopaminergic neurons and chromaffin granules: a mechanism of action.

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Weak base model of amphetamine action

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Amphetamine and other weak bases act to promote reverse transport of dopamine in ventral midbrain neurons.

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Identifiers

DOI

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16 February 2018

PubMed ID

8985716

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16 February 2018