Morphine-induced increase in D-1 receptor regulated signal transduction in rat striatal neurons and its facilitation by glucocorticoid receptor activation: possible role in behavioral sensitization. (Q48873783)

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English	Morphine-induced increase in D-1 receptor regulated signal transduction in rat striatal neurons and its facilitation by glucocorticoid receptor activation: possible role in behavioral sensitization.	scientific article

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scholarly article

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Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8947932

Morphine-induced increase in D-1 receptor regulated signal transduction in rat striatal neurons and its facilitation by glucocorticoid receptor activation: possible role in behavioral sensitization. (English)

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16 February 2018

http://europepmc.org/abstract/MED/8947932

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author name string

A N Schoffelmeer

1

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http://europepmc.org/abstract/MED/8947932

P Voorn

2

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http://europepmc.org/abstract/MED/8947932

A J Jonker

3

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http://europepmc.org/abstract/MED/8947932

G Wardeh

4

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http://europepmc.org/abstract/MED/8947932

P Nestby

5

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http://europepmc.org/abstract/MED/8947932

L J Vanderschuren

6

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T J De Vries

7

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A H Mulder

8

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G H Tjon

9

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publication date

1 November 1996

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Neurochemical Research

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21

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11

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1417-1423

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http://europepmc.org/abstract/MED/8947932

https://scigraph.springernature.com/pub.10.1007/bf02532383

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Stress- and pharmacologically-induced behavioral sensitization increases vulnerability to acquisition of amphetamine self-administration

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The neural basis of drug craving: an incentive-sensitization theory of addiction

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Persistence of the ability of amphetamine preexposure to facilitate acquisition of cocaine self-administration.

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Morphine induces c-fos and junB in striatum and nucleus accumbens via D1 and N-methyl-D-aspartate receptors

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Induction of a long-lasting AP-1 complex composed of altered Fos-like proteins in brain by chronic cocaine and other chronic treatments.

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Neuronal adaptation to amphetamine and dopamine: molecular mechanisms of prodynorphin gene regulation in rat striatum

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Repeated and chronic morphine administration causes differential long-lasting changes in dopaminergic neurotransmission in rat striatum without changing its delta- and kappa-opioid receptor regulation

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Endocrine actions of opioids

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Cocaine stimulates adrenocorticotropin (ACTH) secretion through a corticotropin-releasing factor (CRF)-mediated mechanism.

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A molecular framework for the actions of glucocorticoid hormones in the nervous system

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Sensitization to the psychomotor effects of amphetamine and morphine induced by food restriction depends on corticosterone secretion.

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Inhibition of corticosterone synthesis by Metyrapone decreases cocaine-induced locomotion and relapse of cocaine self-administration.

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Interacting presynaptic kappa-opioid and GABAA receptors modulate dopamine release from rat striatal synaptosomes

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Cellular substrates for interactions between dynorphin terminals and dopamine dendrites in rat ventral tegmental area and substantia nigra.

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The effects of opioid peptides on dopamine release in the nucleus accumbens: an in vivo microdialysis study

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Reciprocal effects of chronic morphine administration on stimulatory and inhibitory G-protein α subunits in primary cultures of rat striatal neurons

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Adaptive changes in the number of Gs- and Gi-proteins underlie adenylyl cyclase sensitization in morphine-treated rat striatal neurons.

1 reference

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21 January 2018

Ontogeny of behavioral sensitization in the rat: effects of direct and indirect dopamine agonists

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Corticosterone differentially regulates the expression of Gs alpha and Gi alpha messenger RNA and protein in rat cerebral cortex

1 reference

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Stress-induced sensitization and glucocorticoids. I. Sensitization of dopamine-dependent locomotor effects of amphetamine and morphine depends on stress-induced corticosterone secretion.

1 reference

https://pubmed.ncbi.nlm.nih.gov/8947932

12 December 2020

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Time course of extracellular dopamine and behavioral sensitization to cocaine. I. Dopamine axon terminals

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https://pubmed.ncbi.nlm.nih.gov/8947932

12 December 2020

based on heuristic

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Time course of extracellular dopamine and behavioral sensitization to cocaine. II. Dopamine perikarya.

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https://pubmed.ncbi.nlm.nih.gov/8947932

12 December 2020

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Amphetamine regulates gene expression in rat striatum via transcription factor CREB.

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https://pubmed.ncbi.nlm.nih.gov/8947932

12 December 2020

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Glucocorticoid receptor activation potentiates the morphine-induced adaptive increase in dopamine D-1 receptor efficacy in gamma-aminobutyric acid neurons of rat striatum/nucleus accumbens

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https://pubmed.ncbi.nlm.nih.gov/8947932

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Mu- and delta-opioid receptors inhibitorily linked to dopamine-sensitive adenylate cyclase in rat striatum display a selectivity profile toward endogenous opioid peptides different from that of presynaptic mu, delta and kappa receptors

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https://pubmed.ncbi.nlm.nih.gov/8947932

12 December 2020

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Mifepristone prevents the expression of long-term behavioural sensitization to amphetamine

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https://pubmed.ncbi.nlm.nih.gov/8947932

12 December 2020

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Pituitary-adrenal axis responses to acute amphetamine in the rat

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https://pubmed.ncbi.nlm.nih.gov/8947932

12 December 2020

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Repeated amphetamine administration induces a prolonged augmentation of phosphorylated cyclase response element-binding protein and Fos-related antigen immunoreactivity in rat striatum

1 reference

https://pubmed.ncbi.nlm.nih.gov/8947932

12 December 2020

based on heuristic

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Regulation of somatodendritic dopamine release in the ventral tegmental area by opioids and GABA: an in vivo microdialysis study

1 reference

https://pubmed.ncbi.nlm.nih.gov/8947932

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Modulation of the locomotor response to amphetamine by corticosterone

1 reference

https://pubmed.ncbi.nlm.nih.gov/8947932

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Molecular aspects of neuropeptide regulation and function in the corpus striatum and nucleus accumbens

1 reference

https://pubmed.ncbi.nlm.nih.gov/8947932

12 December 2020

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Dopamine transmission in the initiation and expression of drug- and stress-induced sensitization of motor activity

1 reference

https://pubmed.ncbi.nlm.nih.gov/8947932

12 December 2020

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Chronic activation of mu- and kappa-opioid receptors in cultured catecholaminergic neurons from rat brain causes neuronal supersensitivity without receptor desensitization

1 reference

https://pubmed.ncbi.nlm.nih.gov/8947932

12 December 2020

based on heuristic

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Critical role for glucocorticoid receptors in stress- and ethanol-induced locomotor sensitization

1 reference

https://pubmed.ncbi.nlm.nih.gov/8947932

12 December 2020

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MK-801 prevents alterations in the mesoaccumbens dopamine system associated with behavioral sensitization to amphetamine.

1 reference

https://pubmed.ncbi.nlm.nih.gov/8947932

12 December 2020

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A rostrocaudal gradient in the synthesis of enkephalin in nucleus accumbens

1 reference

https://pubmed.ncbi.nlm.nih.gov/8947932

12 December 2020

based on heuristic

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Stress-induced sensitization and glucocorticoids. II. Sensitization of the increase in extracellular dopamine induced by cocaine depends on stress-induced corticosterone secretion.

1 reference

https://pubmed.ncbi.nlm.nih.gov/8947932

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Repeated cocaine administration causes persistent enhancement of D1 dopamine receptor sensitivity within the rat nucleus accumbens.

1 reference

https://pubmed.ncbi.nlm.nih.gov/8947932

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1007/BF02532383

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8947932

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8947932

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