Cerebellar long-term depression is deficient in Niemann-Pick type C disease mice. (Q48960390)

17 February 2018

title

Cerebellar long-term depression is deficient in Niemann-Pick type C disease mice. (English)

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17 February 2018

author name string

Cheng-Long Sun

1

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17 February 2018

Li-Da Su

2

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17 February 2018

Qiong Li

3

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17 February 2018

Xin-Xin Wang

4

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17 February 2018

Ying Shen

5

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17 February 2018

publication date

1 March 2011

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17 February 2018

published in

The Cerebellum

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17 February 2018

volume

10

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17 February 2018

issue

1

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17 February 2018

page(s)

88-95

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17 February 2018

https://scigraph.springernature.com/pub.10.1007/s12311-010-0233-2

exact match

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cites work

Niemann-Pick disease type C.

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A defect in cholesterol esterification in Niemann-Pick disease (type C) patients

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Cerebellar degeneration in the Niemann-Pick type C mouse.

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A C57BL/KsJ mouse model of Niemann-Pick disease (spm) belongs to the same complementation group as the major childhood type of Niemann-Pick disease type C.

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Degeneration of neurons and glia in the Niemann-Pick C mouse is unrelated to the low-density lipoprotein receptor

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Cognitive deficit and development of motor impairment in a mouse model of Niemann-Pick type C disease

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Increased activity and altered subcellular distribution of lysosomal enzymes determine neuronal vulnerability in Niemann-Pick type C1-deficient mice

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Niemann-Pick C1 disease gene: homology to mediators of cholesterol homeostasis

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Identification of HE1 as the second gene of Niemann-Pick C disease

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Localization of Niemann-Pick C1 protein in astrocytes: implications for neuronal degeneration in Niemann- Pick type C disease

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Regulated ATP release from astrocytes through lysosome exocytosis.

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Control of basal extracellular adenosine concentration in rat cerebellum.

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Targeted in vivo mutations of the AMPA receptor subunit GluR2 and its interacting protein PICK1 eliminate cerebellar long-term depression.

21 January 2018

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SRF mediates activity-induced gene expression and synaptic plasticity but not neuronal viability

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Adenosine selectively blocks parallel-fiber-mediated synaptic potentials in rat cerebellar cortex

21 January 2018

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Presynaptic inhibition caused by retrograde signal from metabotropic glutamate to cannabinoid receptors

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ATP released by astrocytes mediates glutamatergic activity-dependent heterosynaptic suppression

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Astrocytic purinergic signaling coordinates synaptic networks

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Embryonic Striatal Neurons from Niemann-Pick Type C Mice Exhibit Defects in Cholesterol Metabolism and Neurotrophin Responsiveness

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Before the loss: neuronal dysfunction in Niemann-Pick Type C disease

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12 December 2020

Distinct Localization of P2X receptors at excitatory postsynaptic specializations

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12 December 2020

Decreased purinergic inhibition of synaptic activity in a mouse model of Niemann-Pick disease type C.

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12 December 2020

Contributions of calcium-dependent and calcium-independent mechanisms to presynaptic inhibition at a cerebellar synapse.

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Cerebellar involvement in murine sphingomyelinosis: a new model of Niemann-Pick disease

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12 December 2020

Adenine nucleotides undergo rapid, quantitative conversion to adenosine in the extracellular space in rat hippocampus.

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12 December 2020

Ethanol acutely modulates mGluR1-dependent long-term depression in cerebellum

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12 December 2020

25-Hydroxycholesterol induces reorganization of lysosomes in normal but not Niemann-Pick disease type C astrocytes

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12 December 2020

Pre- and postsynaptic determinants of EPSC waveform at cerebellar climbing fiber and parallel fiber to Purkinje cell synapses.

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Decreased estradiol release from astrocytes contributes to the neurodegeneration in a mouse model of Niemann-Pick disease type C

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12 December 2020

10.1007/S12311-010-0233-2

Identifiers

DOI

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17 February 2018

PubMed ID

21086197

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17 February 2018