Reduced phospholipid breakdown in Alzheimer's brains: a 31P spectroscopy study. (Q49068810)

18 February 2018

title

Reduced phospholipid breakdown in Alzheimer's brains: a 31P spectroscopy study. (English)

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7

Wagner F Gattaz

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author name string

Orestes V Forlenza

1

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Priscilla Wacker

2

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Paula V Nunes

3

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Juliana Yacubian

4

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18 February 2018

Claudio C Castro

5

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Maria C G Otaduy

6

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18 February 2018

publication date

8 February 2005

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18 February 2018

number of pages

7

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inferred from page(s)

published in

Psychopharmacology

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18 February 2018

volume

180

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issue

2

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page(s)

359-365

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18 February 2018

https://scigraph.springernature.com/pub.10.1007/s00213-005-2168-8

exact match

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cites work

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In vivo 31P NMR profiles of Alzheimer's disease and multiple subcortical infarct dementia.

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Altered brain energy metabolism in demented patients with multiple subcortical ischemic lesions. Working hypotheses.

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An in vivo study of phosphorus and glucose metabolism in Alzheimer's disease using magnetic resonance spectroscopy and PET.

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Correlation of phosphorus-31 magnetic resonance spectroscopy and morphologic findings in Alzheimer's disease

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Calcium-sensitive cytosolic phospholipase A2 (cPLA2) is expressed in human brain astrocytes

21 January 2018

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The association between quantitative measures of dementia and of senile change in the cerebral grey matter of elderly subjects.

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Phospholipase A2 activation influences the processing and secretion of the amyloid precursor protein

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A frontal variant of Alzheimer's disease exhibits decreased calcium-independent phospholipase A2 activity in the prefrontal cortex

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Platelet phospholipase A(2) activity in Alzheimer's disease and mild cognitive impairment.

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Neuroinflammatory signaling upregulation in Alzheimer's disease

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Phospholipid-metabolizing enzymes in Alzheimer's disease: increased lysophospholipid acyltransferase activity and decreased phospholipase A2 activity.

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Changes in brain membrane phospholipid and high-energy phosphate metabolism precede dementia

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10.1007/S00213-005-2168-8

Identifiers

DOI

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18 February 2018

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18 February 2018