Cholecalciferol Additively Reduces Serum Parathyroid Hormone Levels in Severe Secondary Hyperparathyroidism Treated with Calcitriol and Cinacalcet among Hemodialysis Patients. (Q50034239)

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scientific article published on 10 February 2018

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English	Cholecalciferol Additively Reduces Serum Parathyroid Hormone Levels in Severe Secondary Hyperparathyroidism Treated with Calcitriol and Cinacalcet among Hemodialysis Patients.	scientific article published on 10 February 2018

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scholarly article

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16 March 2020

Cholecalciferol Additively Reduces Serum Parathyroid Hormone Levels in Severe Secondary Hyperparathyroidism Treated with Calcitriol and Cinacalcet among Hemodialysis Patients (English)

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16 March 2020

hyperparathyroidism

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based on heuristic

inferred from title

secondary hyperparathyroidism

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based on heuristic

inferred from title

cholecalciferol

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based on heuristic

inferred from title

2

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author name string

Cai-Mei Zheng

1

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16 March 2020

Chi-Feng Hung

3

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16 March 2020

Min-Tser Liao

4

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Jia-Fwu Shyu

5

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16 March 2020

Yung-Ho Hsu

6

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16 March 2020

Chien-Lin Lu

7

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Yuan-Hung Wang

8

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Jing-Quan Zheng

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16 March 2020

Tian-Jong Chang

10

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16 March 2020

Kuo-Cheng Lu

12

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publication date

10 February 2018

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16 March 2020

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16 March 2020

10

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16 March 2020

2

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16 March 2020

copyright license

Creative Commons Attribution 4.0 International

10 February 2018

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April 2022 Public Data File from Crossref

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0 references

Cholecalciferol Additively Reduces Serum Parathyroid Hormone and Increases Vitamin D and Cathelicidin Levels in Paricalcitol-Treated Secondary Hyperparathyroid Hemodialysis Patients

1 reference

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Pleiotropic effects of vitamin D in chronic kidney disease

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18 June 2018

Bone histomorphometry before and after long-term treatment with cinacalcet in dialysis patients with secondary hyperparathyroidism

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A randomized controlled trial of cholecalciferol supplementation in patients on maintenance hemodialysis

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Prevalence of 25(OH) vitamin D insufficiency and deficiency in pediatric patients on chronic dialysis

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Regulation of mouse Cyp24a1 expression via promoter-proximal and downstream-distal enhancers highlights new concepts of 1,25-dihydroxyvitamin D(3) action

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Cinacalcet HCl and concurrent low-dose vitamin D improves treatment of secondary hyperparathyroidism in dialysis patients compared with vitamin D alone: the ACHIEVE study results

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Does vitamin D receptor and calcium receptor activation therapy play a role in the histopathologic alterations of parathyroid glands in refractory uremic hyperparathyroidism?

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The OPTIMA study: assessing a new cinacalcet (Sensipar/Mimpara) treatment algorithm for secondary hyperparathyroidism

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Effects of cinacalcet on bone mineral density in patients with secondary hyperparathyroidism

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18 June 2018

Increased 25-hydroxyvitamin D3 1alpha-hydroxylase and reduced 25-hydroxyvitamin D3 24-hydroxylase expression in parathyroid tumors--new prospects for treatment of hyperparathyroidism with vitamin d.

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Extrarenal expression of 25-hydroxyvitamin d(3)-1 alpha-hydroxylase

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Extrarenal production of calcitriol

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Mechanisms through which parathyroid hormone mediates its deleterious effects on organ function in uremia.

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Evidence that 1,25-dihydroxyvitamin D3 inhibits the hepatic production of 25-hydroxyvitamin D in man

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The parathyroid oxyphil cells

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The Effect of Vitamin D Supplementation on Bone Metabolic Markers in Chronic Kidney Disease.

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Role of local versus systemic vitamin D receptors in vascular calcification

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Cholecalciferol in haemodialysis patients: a randomized, double-blind, proof-of-concept and safety study

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14 August 2018

Effects of cinacalcet on bone mineral density and bone markers in hemodialysis patients with secondary hyperparathyroidism

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Direct upregulation of parathyroid calcium-sensing receptor and vitamin D receptor by calcimimetics in uremic rats

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An assessment of cinacalcet HCl effects on bone histology in dialysis patients with secondary hyperparathyroidism.

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Combined therapy with cinacalcet and low doses of vitamin D sterols in patients with moderate to severe secondary hyperparathyroidism

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K/DOQI-recommended intact PTH levels do not prevent low-turnover bone disease in hemodialysis patients.

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Activation of calcium-sensing receptor accelerates apoptosis in hyperplastic parathyroid cells.

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Cinacalcet hydrochloride (Sensipar) in hemodialysis patients on active vitamin D derivatives with controlled PTH and elevated calcium x phosphate

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25-Hydroxyvitamin D(3) suppresses PTH synthesis and secretion by bovine parathyroid cells.

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Achieving NKF-K/DOQI bone metabolism and disease treatment goals with cinacalcet HCl.

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14 August 2018

Calcimimetic compound upregulates decreased calcium-sensing receptor expression level in parathyroid glands of rats with chronic renal insufficiency.

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Low levels of 25-hydroxy vitamin D are associated with elevated parathyroid hormone in healthy adolescent females.

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Cinacalcet for secondary hyperparathyroidism in patients receiving hemodialysis

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14 August 2018

Calcitriol pulse therapy and histology of parathyroid glands in hemodialysis patients

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14 August 2018

Age-related changes in the 25-hydroxyvitamin D versus parathyroid hormone relationship suggest a different reason why older adults require more vitamin D.

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Clinical epidemiology of cardiovascular disease in chronic renal disease

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14 August 2018

Prevalence of abnormal serum vitamin D, PTH, calcium, and phosphorus in patients with chronic kidney disease: results of the study to evaluate early kidney disease

1 reference

https://pubmed.ncbi.nlm.nih.gov/29439405

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Can calcimimetics inhibit parathyroid hyperplasia? Evidence from preclinical studies

1 reference

https://pubmed.ncbi.nlm.nih.gov/29439405

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Histopathological alterations of the parathyroid glands in haemodialysis patients with secondary hyperparathyroidism refractory to cinacalcet hydrochloride

1 reference

https://pubmed.ncbi.nlm.nih.gov/29439405

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Parathyroidectomy for the attainment of NKF-K/DOQI™ and KDIGO recommended values for bone and mineral metabolism in dialysis patients with uncontrollable secondary hyperparathyroidism

1 reference

https://pubmed.ncbi.nlm.nih.gov/29439405

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.3390/NU10020196

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16 March 2020

1 reference

Europe PubMed Central

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16 March 2020

1 reference

Europe PubMed Central

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16 March 2020

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