Advanced glycation end products induce lipogenesis: regulation by natural xanthone through inhibition of ERK and NF-κB. (Q50473781)

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title

Advanced glycation end products induce lipogenesis: regulation by natural xanthone through inhibition of ERK and NF-κB (English)

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1

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Neeharika Verma

series ordinal

2

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author name string

Sunil K Manna

series ordinal

3

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publication date

1 December 2014

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Journal of Cellular Physiology

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published in

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volume

229

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issue

12

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page(s)

1972-1980

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Conditional disruption of the peroxisome proliferator-activated receptor gamma gene in mice results in lowered expression of ABCA1, ABCG1, and apoE in macrophages and reduced cholesterol efflux.

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cites work

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Glyoxal and methylglyoxal levels in diabetic patients: quantitative determination by a new GC/MS method

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Inflammatory stress exacerbates lipid accumulation in hepatic cells and fatty livers of apolipoprotein E knockout mice

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Beta-D-glucoside protects against advanced glycation end products (AGEs)-mediated diabetic responses by suppressing ERK and inducing PPAR gamma DNA binding

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Advanced glycation end products (AGEs) induce apoptosis via a novel pathway: involvement of Ca2+ mediated by interleukin-8 protein.

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Inhibiting TRAF2-mediated activation of NF-kappaB facilitates induction of AP-1.

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Long term environmental tobacco smoke activates nuclear transcription factor-kappa B, activator protein-1, and stress responsive kinases in mouse brain

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Effect of mangiferin on hyperglycemia and atherogenicity in streptozotocin diabetic rats

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Mangiferin decreases plasma free fatty acids through promoting its catabolism in liver by activation of AMPK

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Advanced glycation endproducts interacting with their endothelial receptor induce expression of vascular cell adhesion molecule-1 (VCAM-1) in cultured human endothelial cells and in mice. A potential mechanism for the accelerated vasculopathy of dia

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Advanced glycation end-products: a review

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Hepatocyte-specific IKK-β activation enhances VLDL-triglyceride production in APOE*3-Leiden mice

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High glucose promotes intracellular lipid accumulation in vascular smooth muscle cells by impairing cholesterol influx and efflux balance

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Advanced glycation end products (AGEs) and their receptor (RAGE) system in diabetic retinopathy

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Oxysterol binding protein induces upregulation of SREBP-1c and enhances hepatic lipogenesis.

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Advanced glycation end products (AGEs) increase human mesangial foam cell formation by increasing Golgi SCAP glycosylation in vitro.

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Liquid Chromatographic Determination of Glyoxal and Methylglyoxal from Serum of Diabetic Patients using Meso‐Stilbenediamine as Derivatizing Reagent

21 January 2018

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21 January 2018

Identifiers

DOI

10.1002/JCP.24647

1 reference

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