Role of endothelium in thapsigargin-induced arterial responses in rat aorta. (Q50513079)

14 March 2018

title

Role of endothelium in thapsigargin-induced arterial responses in rat aorta. (English)

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14 March 2018

main subject

endothelium

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author

Francis Ka-Leung Chan

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ORCID Public Data File 2021

author name string

Huang Y

1

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14 March 2018

Yao X

2

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14 March 2018

Lau C

3

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14 March 2018

Chan FL

4

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14 March 2018

Chan NW

5

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14 March 2018

Cheng Y

6

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14 March 2018

Chen Z

7

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14 March 2018

language of work or name

English

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publication date

1 March 2000

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14 March 2018

European Journal of Pharmacology

published in

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14 March 2018

volume

392

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14 March 2018

issue

1-2

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14 March 2018

page(s)

51-59

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14 March 2018

Thapsigargin inhibits the response to acetylcholine and substance P but does not interfere with the responses to endothelium-independent agents

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Thapsigargin discharges intracellular calcium stores and induces transmembrane currents in human endothelial cells

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A novel tumour promoter, thapsigargin, transiently increases cytoplasmic free Ca2+ without generation of inositol phosphates in NG115-401L neuronal cells

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Selective inhibition of agonist-induced but not shear stress-dependent release of endothelial autacoids by thapsigargin

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Thapsigargin, a tumor promoter, discharges intracellular Ca2+ stores by specific inhibition of the endoplasmic reticulum Ca2(+)-ATPase

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The calcium mobilizing tumor promoting agent, thapsigargin elevates the platelet cytoplasmic free calcium concentration to a higher steady state level. A possible mechanism of action for the tumor promotion

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Role of intracellular Ca2+ in EDRF release in rat aorta

7 January 2021

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10.1016/S0014-2999(00)00074-1

7 January 2021

Identifiers

DOI

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14 March 2018

1 reference

14 March 2018