Distinct glycolysis inhibitors determine retinal cell sensitivity to glutamate-mediated injury. (Q50519182)

14 March 2018

title

Distinct glycolysis inhibitors determine retinal cell sensitivity to glutamate-mediated injury. (English)

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14 March 2018

author

Catarina R. Oliveira

4

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14 March 2018

Maria Sancha Santos

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ORCID Public Data File 2021

author name string

A C Rego

1

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14 March 2018

F M Areias

2

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14 March 2018

M S Santos

3

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14 March 2018

publication date

1 March 1999

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14 March 2018

published in

Neurochemical Research

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14 March 2018

volume

24

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14 March 2018

issue

3

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14 March 2018

page(s)

351-358

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14 March 2018

Failure to maintain glycolysis in anoxic nerve terminals

cites work

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Suppression of Mitochondrial Succinate Dehydrogenase, a Primary Target of β‐Amyloid, and Its Derivative Racemized at Ser Residue

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STUDIES ON SUCCINATE-TETRAZOLIUM REDUCTASE SYSTEMS. III. POINTS OF COUPLING OF FOUR DIFFERENT TETRAZOLIUM SALTS

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Oxidative stress, hypoxia, and ischemia-like conditions increase the release of endogenous amino acids by distinct mechanisms in cultured retinal cells

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Inhibition of glutamate uptake induces progressive accumulation of extracellular glutamate and neuronal damage in rat cortical cultures

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beta-Amyloid-mediated inhibition of redox activity (MTT reduction) is not an indicator of astroglial degeneration.

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Identifiers

DOI

10.1023/A:1020977331372

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14 March 2018

Dimensions Publication ID

1005465176

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14 March 2018