Differentiated mTOR but not AMPK signaling after strength vs endurance exercise in training-accustomed individuals. (Q50877255)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:23802289%20AND%20SRC:MED&resulttype=core&format=json

3 March 2020

title

Differentiated mTOR but not AMPK signaling after strength vs endurance exercise in training-accustomed individuals (English)

1 reference

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3 March 2020

2

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3 March 2020

Niels Jessen

6

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3 March 2020

Jean Farup

3

1 reference

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3 March 2020

Tue Kjølhede

4

1 reference

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3 March 2020

author name string

K Vissing

1

1 reference

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3 March 2020

M H Vendelbo

5

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3 March 2020

publication date

1 June 2013

1 reference

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Scandinavian Journal of Medicine and Science in Sports

3 March 2020

published in

1 reference

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3 March 2020

volume

23

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3 March 2020

page(s)

355-366

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3 March 2020

issue

3

1 reference

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Early time course of Akt phosphorylation after endurance and resistance exercise

3 March 2020

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A phosphatidylinositol 3-kinase/protein kinase B-independent activation of mammalian target of rapamycin signaling is sufficient to induce skeletal muscle hypertrophy

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AMPK phosphorylation of raptor mediates a metabolic checkpoint

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A limited role for PI(3,4,5)P3 regulation in controlling skeletal muscle mass in response to resistance exercise.

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Contraction intensity and feeding affect collagen and myofibrillar protein synthesis rates differently in human skeletal muscle.

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Mechanical stimuli regulate rapamycin-sensitive signalling by a phosphoinositide 3-kinase-, protein kinase B- and growth factor-independent mechanism

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Resistance exercise with whey protein ingestion affects mTOR signaling pathway and myostatin in men.

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AMP-activated protein kinase (AMPK) action in skeletal muscle via direct phosphorylation of PGC-1alpha

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Enhanced rates of muscle protein synthesis and elevated mTOR signalling following endurance exercise in human subjects.

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AMPK-mediated regulation of transcription in skeletal muscle.

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Histone modifications and skeletal muscle metabolic gene expression.

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Physiological elevation of endogenous hormones results in superior strength training adaptation.

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Signaling in muscle atrophy and hypertrophy

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Effects of elevated circulating hormones on resistance exercise-induced Akt signaling

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Resistance exercise biology: manipulation of resistance exercise programme variables determines the responses of cellular and molecular signalling pathways

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AMP-activated protein kinase phosphorylates transcription factors of the CREB family

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Muscle Adaptations to Plyometric vs. Resistance Training in Untrained Young Men

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Elevations in ostensibly anabolic hormones with resistance exercise enhance neither training-induced muscle hypertrophy nor strength of the elbow flexors

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5'AMP activated protein kinase expression in human skeletal muscle: effects of strength training and type 2 diabetes.

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Exercise-Induced Phosphorylation of the Novel Akt Substrates AS160 and Filamin A in Human Skeletal Muscle

21 January 2018

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