Genetic engineering cures mice of neurological deficits: prospects for treating Angelman syndrome. (Q50983366)

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31 December 2019

editorial

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title

Genetic engineering cures mice of neurological deficits: prospects for treating Angelman syndrome (English)

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1

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author name string

Ype Elgersma

series ordinal

1

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26 March 2018

http://europepmc.org/abstract/MED/17559342

publication date

1 June 2007

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volume

8

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issue

6

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31 December 2019

page(s)

539-541

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The HMG-CoA reductase inhibitor lovastatin reverses the learning and attention deficits in a mouse model of neurofibromatosis type 1

31 December 2019

cites work

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Suppression of two major Fragile X Syndrome mouse model phenotypes by the mGluR5 antagonist MPEP.

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Reversal of neurological defects in a mouse model of Rett syndrome

21 January 2018

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Rescue of neurological deficits in a mouse model for Angelman syndrome by reduction of alphaCaMKII inhibitory phosphorylation.

21 January 2018

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Neurological aspects of the Angelman syndrome

21 January 2018

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Imprinted expression of the murine Angelman syndrome gene, Ube3a, in hippocampal and Purkinje neurons

21 January 2018

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Mutation of the Angelman ubiquitin ligase in mice causes increased cytoplasmic p53 and deficits of contextual learning and long-term potentiation.

21 January 2018

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Mouse genetic approaches to investigating calcium/calmodulin-dependent protein kinase II function in plasticity and cognition.

21 January 2018

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Calcium/calmodulin-dependent protein kinase II and synaptic plasticity

21 January 2018

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Inhibitory autophosphorylation of CaMKII controls PSD association, plasticity, and learning.

21 January 2018

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Clinical research on Angelman syndrome in the United Kingdom: Observations on 82 affected individuals

21 January 2018

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A previously unrecognised phenotype characterised by obesity, muscular hypotonia, and ability to speak in patients with Angelman syndrome caused by an imprinting defect

21 January 2018

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21 January 2018

Identifiers

DOI

10.2217/14622416.8.6.539

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31 December 2019

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