Complement activation in pauci-immune necrotizing and crescentic glomerulonephritis: results of a proteomic analysis. (Q51066107)

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17 March 2020

title

Complement activation in pauci-immune necrotizing and crescentic glomerulonephritis: results of a proteomic analysis (English)

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17 March 2020

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crescentic glomerulonephritis

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3

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17 March 2020

author name string

Sanjeev Sethi

1

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17 March 2020

Ladan Zand

2

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17 March 2020

Ulrich Specks

4

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17 March 2020

Julie A Vrana

5

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17 March 2020

Siddak Kanwar

6

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17 March 2020

Paul Kurtin

7

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17 March 2020

Jason D Theis

8

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17 March 2020

Andrea Angioi

9

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17 March 2020

Lynn Cornell

10

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17 March 2020

Fernando C Fervenza

11

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17 March 2020

language of work or name

English

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publication date

1 January 2017

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Nephrology Dialysis Transplantation

17 March 2020

published in

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17 March 2020

volume

32

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issue

suppl_1

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17 March 2020

page(s)

i139-i145

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2012 revised International Chapel Hill Consensus Conference Nomenclature of Vasculitides

17 March 2020

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Alternative complement pathway activation products in urine and kidneys of patients with ANCA-associated GN

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Clinical and histological outcome predictors in renal limited pauci-immune crescentic glomerulonephritis: a single centre experience.

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C4d as a Diagnostic Tool in Proliferative GN.

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C5a and its receptors in human anti-neutrophil cytoplasmic antibody (ANCA)-associated vasculitis

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Alternative complement pathway in the pathogenesis of disease mediated by anti-neutrophil cytoplasmic autoantibodies

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C5a receptor (CD88) blockade protects against MPO-ANCA GN

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C5a receptor mediates neutrophil activation and ANCA-induced glomerulonephritis

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ANCA-negative pauci-immune crescentic glomerulonephritis

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ANCA-negative pauci-immune renal vasculitis: histology and outcome

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Differential deposition of C4d and MBL in glomeruli of patients with ANCA-negative pauci-immune crescentic glomerulonephritis.

21 January 2018

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Genetically distinct subsets within ANCA-associated vasculitis

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Revisiting the classification of clinical phenotypes of anti-neutrophil cytoplasmic antibody-associated vasculitis: a cluster analysis.

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Crescentic glomerulonephritis with a paucity of glomerular immunoglobulin localization.

21 January 2018

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Are antineutrophil cytoplasmic antibody-associated vasculitides pauci-immune?

21 January 2018

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p38MAPK, ERK and PI3K signaling pathways are involved in C5a-primed neutrophils for ANCA-mediated activation

21 January 2018

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Renal neutrophils infiltration in antineutrophil cytoplasmic antibodies-negative pauci-immune crescentic glomerulonephritis.

21 January 2018

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Glomeruli of Dense Deposit Disease contain components of the alternative and terminal complement pathway

21 January 2018

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Association of a novel complement factor H mutation with severe crescentic and necrotizing glomerulonephritis

21 January 2018

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C3 glomerulonephritis: clinicopathological findings, complement abnormalities, glomerular proteomic profile, treatment, and follow-up

21 January 2018

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21 January 2018

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Diagnosis of complement alternative pathway disorders

21 January 2018

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Epitope specificity determines pathogenicity and detectability in ANCA-associated vasculitis

21 January 2018

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Laser microdissection and mass spectrometry-based proteomics aids the diagnosis and typing of renal amyloidosis.

21 January 2018

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Mass spectrometry-based proteomic diagnosis of renal immunoglobulin heavy chain amyloidosis

21 January 2018

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Classification of amyloidosis by laser microdissection and mass spectrometry-based proteomic analysis in clinical biopsy specimens

21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

Identifiers

DOI

10.1093/NDT/GFW299

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:28391334%20AND%20SRC:MED&resulttype=core&format=json

17 March 2020

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