Direct inhibition of c-Jun N-terminal kinase in sympathetic neurones prevents c-jun promoter activation and NGF withdrawal-induced death (Q51069152)

28 March 2018

title

Direct inhibition of c-Jun N-terminal kinase in sympathetic neurones prevents c-jun promoter activation and NGF withdrawal-induced death (English)

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28 March 2018

author

Jonathan Whitfield

2

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28 March 2018

author name string

A Eilers

1

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28 March 2018

B Shah

3

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28 March 2018

C Spadoni

4

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28 March 2018

H Desmond

5

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28 March 2018

J Ham

6

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28 March 2018

language of work or name

English

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publication date

1 March 2001

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28 March 2018

Journal of Neurochemistry

published in

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28 March 2018

volume

76

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issue

5

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28 March 2018

page(s)

1439-1454

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28 March 2018

The jun proto-oncogene is positively autoregulated by its product, Jun/AP-1

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The small GTP-binding protein Cdc42 is required for nerve growth factor withdrawal-induced neuronal death

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Characterization of apoptosis in cultured rat sympathetic neurons after nerve growth factor withdrawal

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Role of apoptosis signal-regulating kinase in regulation of the c-Jun N-terminal kinase pathway and apoptosis in sympathetic neurons

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DNA damaging agents induce expression of Fas ligand and subsequent apoptosis in T lymphocytes via the activation of NF-kappa B and AP-1.

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Inhibitors of protein synthesis and RNA synthesis prevent neuronal death caused by nerve growth factor deprivation

21 January 2018

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Viral proteins E1B19K and p35 protect sympathetic neurons from cell death induced by NGF deprivation

21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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Pyridinyl imidazole inhibitors of p38 mitogen-activated protein kinase bind in the ATP site

21 January 2018

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c-Jun is essential for normal mouse development and hepatogenesis

21 January 2018

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10.1046/J.1471-4159.2001.00150.X

21 January 2018

Identifiers

DOI

1 reference

28 March 2018

1 reference

28 March 2018