Effects of hydrogen sulphide in an experimental model of renal ischaemia-reperfusion injury. (Q51298127)

3 April 2018

title

Effects of hydrogen sulphide in an experimental model of renal ischaemia-reperfusion injury. (English)

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author name string

J P Hunter

1

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M Patel

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R Rose

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K Read

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M L Nicholson

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language of work or name

English

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publication date

1 December 2012

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British Journal of Surgery

published in

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volume

99

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issue

12

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page(s)

1665-1671

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Analysis of factors that affect outcome after transplantation of kidneys donated after cardiac death in the UK: a cohort study.

cites work

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Role of hydrogen sulfide in hepatic ischemia-reperfusion-induced injury in rats

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Hydrogen sulfide protects rat lung from ischemia-reperfusion injury

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Hydrogen sulphide ameliorates ischaemia-reperfusion injury in an experimental model of non-heart-beating donor kidney transplantation

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Effects of hydrogen sulphide on ischaemia-reperfusion injury and ischaemic preconditioning in the isolated, perfused rat heart

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Hydrogen sulphide attenuates renal and cardiac injury after total hepatic ischemia and reperfusion.

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Generation of endogenous hydrogen sulfide by cystathionine gamma-lyase limits renal ischemia/reperfusion injury and dysfunction

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Anti-apoptotic and anti-inflammatory effects of hydrogen sulfide in a rat model of regional myocardial I/R

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Anti-apoptotic action of hydrogen sulfide is associated with early JNK inhibition.

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Hydrogen sulfide attenuates myocardial ischemia-reperfusion injury by preservation of mitochondrial function

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Hydrogen sulfide is a signaling molecule and a cytoprotectant

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The cardioprotective potential of hydrogen sulfide in myocardial ischemia/reperfusion injury (review).

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Erythropoietin regulates apoptosis, inflammation and tissue remodelling via caspase-3 and IL-1β in isolated hemoperfused kidneys.

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Cardioprotective effects of hydrogen sulfide

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Hydrogen sulfide preconditioning or neutrophil depletion attenuates ischemia-reperfusion-induced mitochondrial dysfunction in rat small intestine

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Hydrogen sulfide and ischemia-reperfusion injury

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The vasorelaxant effect of H(2)S as a novel endogenous gaseous K(ATP) channel opener

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Exogenous hydrogen sulfide (H2S) protects against regional myocardial ischemia-reperfusion injury--Evidence for a role of K ATP channels

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H2S contributes to the hepatic arterial buffer response and mediates vasorelaxation of the hepatic artery via activation of K(ATP) channels

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Hydrogen sulfide: an endogenous mediator of resolution of inflammation and injury

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Hydrogen sulfide therapy attenuates the inflammatory response in a porcine model of myocardial ischemia/reperfusion injury.

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21 January 2018

Identifiers

DOI

10.1002/BJS.8956

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3 April 2018

PubMed ID

23132416

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3 April 2018