Sodium homeostasis in rat hippocampal slices during oxygen and glucose deprivation: role of voltage-sensitive sodium channels. (Q51434117)

5 April 2018

title

Sodium homeostasis in rat hippocampal slices during oxygen and glucose deprivation: role of voltage-sensitive sodium channels. (English)

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5 April 2018

author name string

Fung ML

series ordinal

1

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5 April 2018

Croning MD

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2

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Haddad GG

series ordinal

3

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5 April 2018

language of work or name

English

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publication date

1 November 1999

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5 April 2018

published in

Neuroscience Letters

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5 April 2018

volume

275

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issue

1

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page(s)

41-44

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5 April 2018

The effect of blocking sodium influx on anoxic damage in the rat hippocampal slice.

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A voltage-dependent persistent sodium current in mammalian hippocampal neurons

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The importance of sodium for anoxic transmission damage in rat hippocampal slices: mechanisms of protection by lidocaine.

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Removal of extracellular sodium prevents anoxia-induced injury in freshly dissociated rat CA1 hippocampal neurons.

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Mechanisms of anoxia-induced depolarization in brainstem neurons: in vitro current and voltage clamp studies in the adult rat

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Effect of anoxia on ion distribution in the brain

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O2 tension in adult and neonatal brain slices under several experimental conditions

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Cl- and Na+ homeostasis during anoxia in rat hypoglossal neurons: intracellular and extracellular in vitro studies

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The barbiturate thiopental reduces ATP levels during anoxia but improves electrophysiological recovery and ionic homeostasis in the rat hippocampal slice.

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Mechanisms of ischemic brain damage

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Hippocampal slices: glutamate overflow and cellular damage from ischemia are reduced by sodium-channel blockade.

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Na+ channels as targets for neuroprotective drugs

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Mechanisms underlying the rapid depolarization produced by deprivation of oxygen and glucose in rat hippocampal CA1 neurons in vitro

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Damage from oxygen and glucose deprivation in hippocampal slices is prevented by tetrodotoxin, lidocaine and phenytoin without blockade of action potentials.

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7 January 2021

10.1016/S0304-3940(99)00728-4

Identifiers

DOI

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5 April 2018

PubMed ID

10554980

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5 April 2018