Hypoxia paradoxically inhibits the angiogenic response of isolated vessel explants while inducing overexpression of vascular endothelial growth factor. (Q51557632)

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English	Hypoxia paradoxically inhibits the angiogenic response of isolated vessel explants while inducing overexpression of vascular endothelial growth factor.	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

7 April 2018

http://europepmc.org/abstract/MED/26748649

Hypoxia paradoxically inhibits the angiogenic response of isolated vessel explants while inducing overexpression of vascular endothelial growth factor. (English)

1 reference

Europe PubMed Central

PubMed publication ID

7 April 2018

http://europepmc.org/abstract/MED/26748649

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author name string

Alfred C Aplin

1

1 reference

Europe PubMed Central

PubMed publication ID

7 April 2018

http://europepmc.org/abstract/MED/26748649

Roberto F Nicosia

2

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Europe PubMed Central

PubMed publication ID

7 April 2018

http://europepmc.org/abstract/MED/26748649

publication date

9 January 2016

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Europe PubMed Central

PubMed publication ID

7 April 2018

http://europepmc.org/abstract/MED/26748649

1 reference

Europe PubMed Central

PubMed publication ID

7 April 2018

http://europepmc.org/abstract/MED/26748649

19

1 reference

Europe PubMed Central

PubMed publication ID

7 April 2018

http://europepmc.org/abstract/MED/26748649

2

1 reference

Europe PubMed Central

PubMed publication ID

7 April 2018

http://europepmc.org/abstract/MED/26748649

133-146

1 reference

Europe PubMed Central

PubMed publication ID

7 April 2018

http://europepmc.org/abstract/MED/26748649

Role of hypoxia in the evolution and development of the cardiovascular system

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Identifiers

10.1007/S10456-015-9493-2

1 reference

Europe PubMed Central

PubMed publication ID

7 April 2018

http://europepmc.org/abstract/MED/26748649

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

7 April 2018

http://europepmc.org/abstract/MED/26748649

ResearchGate publication ID

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