Genetic inactivation of ApoJ/clusterin: effects on prostate tumourigenesis and metastatic spread. (Q51612614)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19784068%20AND%20SRC:MED&resulttype=core&format=json

8 February 2020

title

Genetic inactivation of ApoJ/clusterin: effects on prostate tumourigenesis and metastatic spread (English)

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8 February 2020

5

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8 February 2020

Matteo Fassan

10

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8 February 2020

Saverio Bettuzzi

1

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8 February 2020

Serena Davoli

3

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8 February 2020

author name string

P Davalli

2

1 reference

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8 February 2020

O Chayka

4

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8 February 2020

L Belloni

6

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8 February 2020

D Pellacani

7

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8 February 2020

G Fregni

8

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8 February 2020

S Astancolle

9

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8 February 2020

A Corti

11

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8 February 2020

R Baffa

12

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8 February 2020

A Sala

13

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8 February 2020

language of work or name

English

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publication date

1 December 2009

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8 February 2020

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8 February 2020

volume

28

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8 February 2020

issue

49

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8 February 2020

page(s)

4344-4352

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Identification of an androgen-repressed mRNA in rat ventral prostate as coding for sulphated glycoprotein 2 by cDNA cloning and sequence analysis

8 February 2020

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NF-kappa B activates prostate-specific antigen expression and is upregulated in androgen-independent prostate cancer

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Clusterin-mediated apoptosis is regulated by adenomatous polyposis coli and is p21 dependent but p53 independent.

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Dissociation of epithelial and neuroendocrine carcinoma lineages in the transgenic adenocarcinoma of mouse prostate model of prostate cancer

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Ram rete testis fluid contains a protein (clusterin) which influences cell-cell interactions in vitro

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Use of antisense oligonucleotides targeting the cytoprotective gene, clusterin, to enhance androgen- and chemo-sensitivity in prostate cancer

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PML targeting eradicates quiescent leukaemia-initiating cells

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Clusterin expression is significantly enhanced in prostate cancer cells following androgen withdrawal therapy

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Apolipoprotein J/clusterin limits the severity of murine autoimmune myocarditis

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E2F1 represses beta-catenin transcription and is antagonized by both pRB and CDK8

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Clusterin is epigenetically regulated in prostate cancer

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Essential requirement of apolipoprotein J (clusterin) signaling for IkappaB expression and regulation of NF-kappaB activity.

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Clusterin (SGP-2, ApoJ) expression is downregulated in low- and high-grade human prostate cancer.

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Challenge and promise: roles for clusterin in pathogenesis, progression and therapy of cancer

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