Hypoxia decreases Runx2/Cbfa1 expression in human osteoblast-like cells. (Q51712401)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12088880%20AND%20SRC:MED&resulttype=core&format=json

10 November 2019

title

Hypoxia decreases Runx2/Cbfa1 expression in human osteoblast-like cells (English)

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10 November 2019

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J H Park

1

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10 November 2019

B H Park

2

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10 November 2019

H K Kim

3

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10 November 2019

T S Park

4

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10 November 2019

H S Baek

5

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10 November 2019

publication date

1 June 2002

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Molecular and Cellular Endocrinology

10 November 2019

published in

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10 November 2019

volume

192

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10 November 2019

issue

1-2

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10 November 2019

page(s)

197-203

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Hypoxia induces vascular endothelial growth factor gene transcription in human osteoblast-like cells through the hypoxia-inducible factor-2alpha

10 November 2019

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Osf2/Cbfa1: a transcriptional activator of osteoblast differentiation

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A Cbfa1-dependent genetic pathway controls bone formation beyond embryonic development

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Collagenase 3 is a target of Cbfa1, a transcription factor of the runt gene family involved in bone formation

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Role of Cbfa1 in osteoblast differentiation and function

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Changes in bone marrow blood flow with aging

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Targeted disruption of Cbfa1 results in a complete lack of bone formation owing to maturational arrest of osteoblasts

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Comparison of the bone mineral content of the lower limbs in men with ischaemic atherosclerotic disease

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Study of the intraosseous vessels of the femoral head in patients with fractures of the femoral neck or osteoarthritis of the hip

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Runx2 is a common target of transforming growth factor beta1 and bone morphogenetic protein 2, and cooperation between Runx2 and Smad5 induces osteoblast-specific gene expression in the pluripotent mesenchymal precursor cell line C2C12.

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7 January 2021

Nitroglycerin therapy is as efficacious as standard estrogen replacement therapy (Premarin) in prevention of oophorectomy-induced bone loss: a human pilot clinical study

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Hypoxia regulates osteoblast gene expression

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7 January 2021

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7 January 2021

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Identifiers

DOI

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10 November 2019

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