Intrauterine programming of bone. Part 1: alteration of the osteogenic environment. (Q51741812)

11 April 2018

title

Intrauterine programming of bone. Part 1: alteration of the osteogenic environment. (English)

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11 April 2018

author

Cyrus Cooper

3

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11 April 2018

Richard Oreffo

4

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11 April 2018

author name string

S A Lanham

1

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11 April 2018

C Roberts

2

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language of work or name

English

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publication date

15 August 2007

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11 April 2018

Osteoporosis International

published in

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11 April 2018

volume

19

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11 April 2018

issue

2

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11 April 2018

page(s)

147-156

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11 April 2018

https://scigraph.springernature.com/pub.10.1007/s00198-007-0443-8

exact match

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cites work

Maternal low-protein diet in rat pregnancy programs blood pressure through sex-specific mechanisms

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Crossref

https://api.crossref.org/works/10.1007%2FS00198-007-0443-8

Dietary protein deficiency induces osteoporosis in aged male rats

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Novel insights into the pathogenesis of osteoporosis: the role of intrauterine programming

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Childhood growth, physical activity, and peak bone mass in women

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Circulating levels of IGF-1 directly regulate bone growth and density

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Sex-specific effects of prenatal low-protein and carbenoxolone exposure on renal angiotensin receptor expression in rats

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Fetal nutrition and adult disease

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Protein undernutrition-induced bone loss is associated with decreased IGF-I levels and estrogen deficiency

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Normal growth and development in the absence of hepatic insulin-like growth factor I

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Physiological mechanisms adopted by chondrocytes in regulating longitudinal bone growth in rats

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Mesenchymal stem cells: lineage, plasticity, and skeletal therapeutic potential

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The fetal origins of diseases of old age.

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Fetal exposure to a maternal low protein diet impairs nephrogenesis and promotes hypertension in the rat

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Dietary restriction in pregnant rats causes gender-related hypertension and vascular dysfunction in offspring

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Programming of hepatic antioxidant capacity and oxidative injury in the ageing rat.

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Intrauterine exposure to a maternal low protein diet reduces adult bone mass and alters growth plate morphology in rats

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Do genetic markers aid in risk assessment?

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The fetal origins of osteoporotic fracture

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Altered regulation of hepatic glucose output in the male offspring of protein-malnourished rat dams

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Dietary protein restriction lowers plasma insulin-like growth factor I (IGF-I), impairs cortical bone formation, and induces osteoblastic resistance to IGF-I in adult female rats.

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Prenatal Exposure to a Maternal Low Protein Diet Shortens Life Span in Rats

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Vitamin D deficiency and osteopathies

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Aspects of the maturation process in growth cartilage in the rat tibia

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Maternal protein deficiency affects mesenchymal stem cell activity in the developing offspring

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Growth in infancy and bone mass in later life

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Weanling rats exposed to maternal low-protein diets during discrete periods of gestation exhibit differing severity of hypertension

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Programming of growth hormone secretion and bone mineral density in elderly men: a hypothesis.

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Bones, muscles and visceral organs of protein-malnourished rats (Rattus norvegicus) grow more slowly but for longer durations to reach normal final size

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Postnatal catch-up growth induced by growth hormone and insulin-like growth factor-I in rats with intrauterine growth retardation caused by maternal protein malnutrition.

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Protein malnutrition affects the growth trajectories of the craniofacial skeleton in rats

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Human bone marrow osteoprogenitors express estrogen receptor-alpha and bone morphogenetic proteins 2 and 4 mRNA during osteoblastic differentiation

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12 December 2020

10.1007/S00198-007-0443-8

Identifiers

DOI

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11 April 2018

PubMed ID

17701093

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11 April 2018