Retinoic Acid Improves Incidence and Severity of Necrotizing Enterocolitis by Lymphocyte Balance Restitution and Repopulation of LGR5+ Intestinal Stem Cells. (Q52856803)

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scientific article published on 2 August 2016

Language	Label	Description	Also known as
English	Retinoic Acid Improves Incidence and Severity of Necrotizing Enterocolitis by Lymphocyte Balance Restitution and Repopulation of LGR5+ Intestinal Stem Cells.	scientific article published on 2 August 2016

Statements

scholarly article

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20 March 2020

Retinoic Acid Improves Incidence and Severity of Necrotizing Enterocolitis by Lymphocyte Balance Restitution and Repopulation of LGR5+ Intestinal Stem Cells (English)

1 reference

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20 March 2020

Lgr5+ intestinal stem-cell

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based on heuristic

inferred from title

necrotizing enterocolitis

1 reference

based on heuristic

inferred from title

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based on heuristic

inferred from title

4

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10

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author name string

Diego F Niño

1

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Chhinder P Sodhi

2

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Charlotte E Egan

3

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Joyce Lin

5

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Peng Lu

6

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Yukihiro Yamaguchi

7

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Hongpeng Jia

8

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Laura Y Martin

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William B Fulton

11

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20 March 2020

Thomas Prindle

12

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20 March 2020

John A Ozolek

13

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David J Hackam

14

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publication date

1 January 2017

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20 March 2020

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20 March 2020

47

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20 March 2020

22-32

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1

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20 March 2020

Necrotizing enterocolitis: new insights into pathogenesis and mechanisms

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Toll-like receptor 4-mediated lymphocyte influx induces neonatal necrotizing enterocolitis.

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Intestinal stem cell growth and differentiation on a tubular scaffold with evaluation in small and large animals

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Trends in Care Practices, Morbidity, and Mortality of Extremely Preterm Neonates, 1993-2012.

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Breast milk protects against the development of necrotizing enterocolitis through inhibition of Toll-like receptor 4 in the intestinal epithelium via activation of the epidermal growth factor receptor

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Outcomes and costs of surgical treatments of necrotizing enterocolitis

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Toll-like receptor 4-mediated endoplasmic reticulum stress in intestinal crypts induces necrotizing enterocolitis.

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Toll-like receptor regulation of intestinal development and inflammation in the pathogenesis of necrotizing enterocolitis

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Adult intestinal stem cells: critical drivers of epithelial homeostasis and regeneration

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Endothelial TLR4 activation impairs intestinal microcirculatory perfusion in necrotizing enterocolitis via eNOS-NO-nitrite signaling

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Toll-like receptor 4 is expressed on intestinal stem cells and regulates their proliferation and apoptosis via the p53 up-regulated modulator of apoptosis

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A nomenclature for intestinal in vitro cultures.

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Necrotising enterocolitis is characterised by disrupted immune regulation and diminished mucosal regulatory (FOXP3)/effector (CD4, CD8) T cell ratios.

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22 May 2018

Toll-like receptor-4 inhibits enterocyte proliferation via impaired beta-catenin signaling in necrotizing enterocolitis

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Single Lgr5 stem cells build crypt-villus structures in vitro without a mesenchymal niche

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Cytokines in necrotizing enterocolitis

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13 August 2018

Necrotizing enterocolitis

1 reference

https://pubmed.ncbi.nlm.nih.gov/27488085

12 December 2020

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Cutting edge: depletion of Foxp3+ cells leads to induction of autoimmunity by specific ablation of regulatory T cells in genetically targeted mice

1 reference

https://pubmed.ncbi.nlm.nih.gov/27488085

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Isolation of mouse lymphocytes from small intestine tissues

1 reference

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Critical role of IL-17 receptor signaling in acute TNBS-induced colitis

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based on heuristic

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Identifiers

10.1097/SHK.0000000000000713

1 reference

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1 reference

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20 March 2020

1 reference

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20 March 2020

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