Mice defective in the DNA mismatch gene PMS2 are hypersensitive to MNU induced thymic lymphoma and are partially protected by transgenic expression of human MGMT. (Q53420486)

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scientific article published in July 1999

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English	Mice defective in the DNA mismatch gene PMS2 are hypersensitive to MNU induced thymic lymphoma and are partially protected by transgenic expression of human MGMT.	scientific article published in July 1999

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scholarly article

1 reference

Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/10439048

Mice defective in the DNA mismatch gene PMS2 are hypersensitive to MNU induced thymic lymphoma and are partially protected by transgenic expression of human MGMT. (English)

1 reference

Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/10439048

author name string

X Qin

1

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Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/10439048

L Liu

2

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Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/10439048

S L Gerson

3

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14 May 2018

http://europepmc.org/abstract/MED/10439048

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publication date

1 July 1999

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Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/10439048

1 reference

Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/10439048

18

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Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/10439048

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Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/10439048

4394-4400

1 reference

Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/10439048

https://scigraph.springernature.com/pub.10.1038/sj.onc.1202798

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1 reference

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Male mice defective in the DNA mismatch repair gene PMS2 exhibit abnormal chromosome synapsis in meiosis

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Involvement of mouse Mlh1 in DNA mismatch repair and meiotic crossing over

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High incidence of nitrosamine-induced tumorigenesis in mice lacking DNA repair methyltransferase.

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DNA Repair Enzymes

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Elevated levels of mutation in multiple tissues of mice deficient in the DNA mismatch repair gene Pms2

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Tumour susceptibility and spontaneous mutation in mice deficient in Mlh1, Pms1 and Pms2 DNA mismatch repair

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MSH2 deficient mice are viable and susceptible to lymphoid tumours

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Influence of sex and carcinogen treatment protocol on tumor latency and frequency of K-ras mutations in N-methyl-N-nitrosourea-induced lymphomas.

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Induction of mammary carcinomas in rats by nitroso-methylurea involves malignant activation of H-ras-1 locus by single point mutations.

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Repair of O6-ethylguanine in DNA protects rat 208F cells from tumorigenic conversion by N-ethyl-N-nitrosourea

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Mechanism of mutagenesis by O6-methylguanine

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The prevention of thymic lymphomas in transgenic mice by human O6-alkylguanine-DNA alkyltransferase

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O6-alkylguanine DNA lesions trigger apoptosis

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DNA adduct formation and assessment of aberrant crypt foci in vivo in the rat colon mucosa after treatment with N-methyl-N-nitrosourea

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Targeted disruption of the DNA repair methyltransferase gene renders mice hypersensitive to alkylating agent

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Transgenic expression of human MGMT protects against azoxymethane-induced aberrant crypt foci and G to A mutations in the K-ras oncogene of mouse colon

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Identifiers

10.1038/SJ.ONC.1202798

1 reference

Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/10439048

1 reference

Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/10439048

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