2,3,7,8-Tetrachlorodibenzo-p-dioxin suppresses apoptosis and leads to hyperphosphorylation of p53 in rat hepatocytes. (Q53428667)

14 May 2018

title

2,3,7,8-Tetrachlorodibenzo-p-dioxin suppresses apoptosis and leads to hyperphosphorylation of p53 in rat hepatocytes. (English)

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14 May 2018

author name string

Wörner W

series ordinal

1

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14 May 2018

Schrenk D

series ordinal

2

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14 May 2018

publication date

1 December 1998

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14 May 2018

Environmental Toxicology and Pharmacology

published in

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14 May 2018

volume

6

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14 May 2018

issue

4

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14 May 2018

page(s)

239-247

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14 May 2018

The liver-specific response to phenobarbital involves a transient increase in phosphorylation of a 34-kda nuclear protein in rat liver and in hepatocytes in culture

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The ability to alter the gap junction protein expression outside GST-P positive foci in liver of rats was associated to the tumour promotion potency of different polychlorinated biphenyls

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Suppression of liver cell apoptosis in vitro by the non-genotoxic hepatocarcinogen and peroxisome proliferator nafenopin

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Controlled death (apoptosis) of normal and putative preneoplastic cells in rat liver following withdrawal of tumor promoters

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Evidence for a second pathway in the action mechanism of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD). Significance of Ah-receptor mediated activation of protein kinase under cell-free conditions

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Identification of c-Src as the integral component of the cytosolic Ah receptor complex, transducing the signal of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) through the protein phosphorylation pathway

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Phosphorylation of p53 at the casein kinase II site selectively regulates p53-dependent transcriptional repression but not transactivation

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p53 tumor suppressor gene: from the basic research laboratory to the clinic--an abridged historical perspective

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Effects of the tumour promoter okadaic acid on intracellular protein phosphorylation and metabolism

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Constitutive phosphorylation of the receptor for insulinlike growth factor I in cells transformed by the src oncogene

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Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4

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Differential induction of transcriptionally active p53 following UV or ionizing radiation: defects in chromosome instability syndromes?

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UV Irradiation Stimulates Levels of p53 Cellular Tumor Antigen in Nontransformed Mouse Cells

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Nuclear protein phosphorylation and growth control

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Role of the Ah receptor and the dioxin-inducible [Ah] gene battery in toxicity, cancer, and signal transduction.

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Induction of apoptosis in cultured hepatocytes and in regressing liver by transforming growth factor beta 1

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2,3,7,8-Tetrachlorodibenzo-p-dioxin and ethinylestradiol as co-mitogens in cultured rat hepatocytes

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Promotion of preneoplastic foci in rat liver with 2,3,7,8-tetrachlorodibenzo-p-dioxin, 1,2,3,4,6,7,8-heptachloro-dibenzo-p-dioxin and a defined mixture of 49 polychlorinated dibenzo-p-dioxins

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Tumor promotion in the liver

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GST-P-positive hepatocytes isolated from rats bearing enzyme-altered foci show no signs of p53 protein induction and replicate even when their DNA contains strand breaks.

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Inhibition of apoptosis during 2,3,7,8-tetrachlorodibenzo-p-dioxin-mediated tumour promotion in rat liver

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Association of insulin-like growth-factor-I-induced DNA synthesis with phosphorylation and nuclear exclusion of p53 in human breast cancer MCF-7 cells

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Identifiers

DOI

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14 May 2018

PubMed ID

21781900

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14 May 2018