Src suppressed C kinase substrate regulates the lipopolysaccharide-induced TNF-alpha biosynthesis in rat astrocytes. (Q53527494)

15 May 2018

title

Src suppressed C kinase substrate regulates the lipopolysaccharide-induced TNF-alpha biosynthesis in rat astrocytes. (English)

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15 May 2018

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Lin-lin Sun

1

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15 May 2018

Chun Cheng

2

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15 May 2018

Hai-ou Liu

3

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15 May 2018

Cong-cong Shen

4

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15 May 2018

Feng Xiao

5

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15 May 2018

Jing Qin

6

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15 May 2018

Jun-ling Yang

7

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15 May 2018

Ai-guo Shen

8

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15 May 2018

publication date

1 January 2007

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15 May 2018

Journal of Molecular Neuroscience

published in

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15 May 2018

volume

32

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15 May 2018

issue

1

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15 May 2018

page(s)

16-24

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15 May 2018

https://scigraph.springernature.com/pub.10.1007/s12031-007-0003-x

exact match

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cites work

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Calmodulin and cyclin D anchoring sites on the Src-suppressed C kinase substrate, SSeCKS.

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Involvement of the protein kinase C substrate, SSeCKS, in the actin-based stellate morphology of mesangial cells

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SSeCKS, a major protein kinase C substrate with tumor suppressor activity, regulates G(1)-->S progression by controlling the expression and cellular compartmentalization of cyclin D.

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Control of cytoskeletal architecture by the src-suppressed C kinase substrate, SSeCKS.

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The p44/42 mitogen-activated protein kinase cascade is involved in the induction and maintenance of astrocyte stellation mediated by protein kinase C

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Tumor necrosis factor-alpha (TNF-alpha) increases both in the brain and in the cerebrospinal fluid from parkinsonian patients

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Lipopolysaccharide stimulates differential expression of myristoylated protein kinase C substrates in murine microglia

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Cell type-specific upregulation of myristoylated alanine-rich C kinase substrate and protein kinase C-alpha, -beta I, -beta II, and -delta in microglia following kainic acid-induced seizures

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Palmitate induces tumor necrosis factor-alpha expression in C2C12 skeletal muscle cells by a mechanism involving protein kinase C and nuclear factor-kappaB activation

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10.1007/S12031-007-0003-X

Identifiers

DOI

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15 May 2018

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15 May 2018