Complement activation promotes colitis-associated carcinogenesis through activating intestinal IL-1β/IL-17A axis. (Q53595887)

16 May 2018

title

Complement activation promotes colitis-associated carcinogenesis through activating intestinal IL-1β/IL-17A axis. (English)

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16 May 2018

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C Ning

1

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16 May 2018

Y-Y Li

2

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16 May 2018

Y Wang

3

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G-C Han

4

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16 May 2018

R-X Wang

5

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H Xiao

6

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16 May 2018

X-Y Li

7

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C-M Hou

8

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Y-F Ma

9

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D-S Sheng

10

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B-F Shen

11

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J-N Feng

12

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16 May 2018

R-F Guo

13

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16 May 2018

Y Li

14

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G-J Chen

15

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16 May 2018

publication date

4 March 2015

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16 May 2018

published in

Mucosal Immunology

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16 May 2018

volume

8

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16 May 2018

issue

6

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16 May 2018

page(s)

1275-1284

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16 May 2018

The risk of colorectal cancer in ulcerative colitis: a meta-analysis

cites work

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Human colonic epithelial cells detect and respond to C5a via apically expressed C5aR through the ERK pathway

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IL-6 and Stat3 are required for survival of intestinal epithelial cells and development of colitis-associated cancer

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IL-1β mediates chronic intestinal inflammation by promoting the accumulation of IL-17A secreting innate lymphoid cells and CD4(+) Th17 cells

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Resveratrol attenuates C5a-induced inflammatory responses in vitro and in vivo by inhibiting phospholipase D and sphingosine kinase activities

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Dextran sulfate sodium-induced colitis occurs in severe combined immunodeficient mice.

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MPO-ANCA induces IL-17 production by activated neutrophils in vitro via classical complement pathway-dependent manner

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IL-1β-induced activation of p38 promotes metastasis in gastric adenocarcinoma via upregulation of AP-1/c-fos, MMP2 and MMP9

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IL-17, produced by lymphocytes and neutrophils, is necessary for lipopolysaccharide-induced airway neutrophilia: IL-15 as a possible trigger.

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12 December 2020

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12 December 2020

Expression of cell membrane complement regulatory glycoproteins along the normal and diseased human gastrointestinal tract

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12 December 2020

Microbiota-induced IL-1β, but not IL-6, is critical for the development of steady-state TH17 cells in the intestine

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12 December 2020

Enhanced secretion of tumour necrosis factor-alpha, IL-6, and IL-1 beta by isolated lamina propria mononuclear cells from patients with ulcerative colitis and Crohn's disease

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12 December 2020

Evidence for anti-inflammatory effects of C5a on the innate IL-17A/IL-23 axis

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12 December 2020

IL-17 produced by neutrophils regulates IFN-gamma-mediated neutrophil migration in mouse kidney ischemia-reperfusion injury

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12 December 2020

CX₃CR1⁺ mononuclear phagocytes support colitis-associated innate lymphoid cell production of IL-22.

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Drusen complement components C3a and C5a promote choroidal neovascularization

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The role of the complement and contact systems in the dextran sulfate sodium-induced colitis model: the effect of C1 inhibitor in inflammatory bowel disease

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Modulation of the antitumor immune response by complement

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Essential roles of IL-6 trans-signaling in colonic epithelial cells, induced by the IL-6/soluble-IL-6 receptor derived from lamina propria macrophages, on the development of colitis-associated premalignant cancer in a murine model

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Anaphylatoxin C5a creates a favorable microenvironment for lung cancer progression.

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12 December 2020

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12 December 2020

Decay-accelerating factor deficiency increases susceptibility to dextran sulfate sodium-induced colitis: role for complement in inflammatory bowel disease

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12 December 2020

A novel method in the induction of reliable experimental acute and chronic ulcerative colitis in mice

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12 December 2020

γδT17 cells promote the accumulation and expansion of myeloid-derived suppressor cells in human colorectal cancer.

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12 December 2020

A human colonic commensal promotes colon tumorigenesis via activation of T helper type 17 T cell responses

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12 December 2020

A potent human C5a receptor antagonist protects against disease pathology in a rat model of inflammatory bowel disease

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12 December 2020

Identifiers

DOI

10.1038/MI.2015.18

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16 May 2018

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16 May 2018