Expression profile of genes related to osteoclastogenesis in mouse growth plate and articular cartilage. (Q53649562)

16 May 2018

title

Expression profile of genes related to osteoclastogenesis in mouse growth plate and articular cartilage. (English)

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16 May 2018

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Kenta Kishimoto

1

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16 May 2018

Riko Kitazawa

2

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16 May 2018

Masahiro Kurosaka

3

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16 May 2018

Sakan Maeda

4

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16 May 2018

Sohei Kitazawa

5

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16 May 2018

publication date

8 November 2005

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16 May 2018

Histochemistry and Cell Biology

published in

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16 May 2018

volume

125

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16 May 2018

issue

5

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16 May 2018

page(s)

593-602

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16 May 2018

https://scigraph.springernature.com/pub.10.1007/s00418-005-0103-z

exact match

0 references

cites work

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Heterodimerization of Msx and Dlx homeoproteins results in functional antagonism

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Promoter structure of mouse RANKL/TRANCE/OPGL/ODF gene

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Targeted disruption of Cbfa1 results in a complete lack of bone formation owing to maturational arrest of osteoblasts

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BMP-2-induced Runx2 expression is mediated by Dlx5, and TGF-beta 1 opposes the BMP-2-induced osteoblast differentiation by suppression of Dlx5 expression

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Induction of Osteoclast Differentiation by Runx2 through Receptor Activator of Nuclear Factor-κB Ligand (RANKL) and Osteoprotegerin Regulation and Partial Rescue of Osteoclastogenesis in Runx2–/– Mice by RANKL Transgene

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Runt homology domain proteins in osteoblast differentiation: AML3/CBFA1 is a major component of a bone-specific complex

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Regulation of chondrocyte differentiation by Cbfa1

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Upstream and downstream targets of RUNX proteins.

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Cbfa1 does not regulate RANKL gene activity in stromal/osteoblastic cells

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12 December 2020

Regulation of the activity of the transcription factor Runx2 by two homeobox proteins, Msx2 and Dlx5

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Vitamin D(3) augments osteoclastogenesis via vitamin D-responsive element of mouse RANKL gene promoter

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RANK ligand, RANK, and OPG expression in type II collagen-induced arthritis mouse

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Osteoclast differentiation and characteristic trabecular bone formation during growth plate destruction in osteoprotegerin-deficient mice.

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Reciprocal regulation of osteocalcin transcription by the homeodomain proteins Msx2 and Dlx5.

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Cbfa1, a candidate gene for cleidocranial dysplasia syndrome, is essential for osteoblast differentiation and bone development

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Skeletal malformations caused by overexpression of Cbfa1 or its dominant negative form in chondrocytes

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A BMP-inducible gene, dlx5, regulates osteoblast differentiation and mesoderm induction

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Msx2 deficiency in mice causes pleiotropic defects in bone growth and ectodermal organ formation

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Impaired vascular invasion of Cbfa1-deficient cartilage engrafted in the spleen

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Bone morphogenetic protein-2-induced alkaline phosphatase expression is stimulated by Dlx5 and repressed by Msx2

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VEGF couples hypertrophic cartilage remodeling, ossification and angiogenesis during endochondral bone formation

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Cooperative interactions between RUNX2 and homeodomain protein-binding sites are critical for the osteoblast-specific expression of the bone sialoprotein gene

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Hedgehog signaling enhances core-binding factor a1 and receptor activator of nuclear factor-kappaB ligand (RANKL) gene expression in chondrocytes

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Heparanase mRNA expression during fracture repair in mice

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Maturational disturbance of chondrocytes in Cbfa1-deficient mice

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Cbfa1/Runx2 gene expression in articular chondrocytes of the mice temporomandibular and knee joints in vivo

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A fundamental transcription factor for bone and cartilage

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In situ hybridization with polymerase chain reaction-derived single-stranded DNA probe and S1 nuclease

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Dlx5 is a positive regulator of chondrocyte differentiation during endochondral ossification

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RUNX transcription factors as key targets of TGF-beta superfamily signaling

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Essential role of beta-catenin in postnatal bone acquisition

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10.1007/S00418-005-0103-Z

Identifiers

DOI

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16 May 2018

PubMed ID

16283360

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16 May 2018