Matrilysin shedding of syndecan-1 regulates chemokine mobilization and transepithelial efflux of neutrophils in acute lung injury. (Q53662097)

17 May 2018

title

Matrilysin shedding of syndecan-1 regulates chemokine mobilization and transepithelial efflux of neutrophils in acute lung injury. (English)

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17 May 2018

author

Pyong Woo Park

2

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17 May 2018

author name string

Qinglang Li

1

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17 May 2018

Carole L Wilson

3

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17 May 2018

William C Parks

4

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17 May 2018

language of work or name

English

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publication date

1 November 2002

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17 May 2018

published in

Cell

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17 May 2018

volume

111

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17 May 2018

issue

5

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17 May 2018

page(s)

635-646

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17 May 2018

Syndecan-1 is required for Wnt-1-induced mammary tumorigenesis in mice

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Shedding of syndecan-1 and -4 ectodomains is regulated by multiple signaling pathways and mediated by a TIMP-3-sensitive metalloproteinase

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Transepithelial neutrophil migration is CXCR1 dependent in vitro and is defective in IL-8 receptor knockout mice

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7 January 2021

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7 January 2021

Intestinal tumorigenesis is suppressed in mice lacking the metalloproteinase matrilysin

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7 January 2021

The metalloproteinase matrilysin is preferentially expressed by epithelial cells in a tissue-restricted pattern in the mouse

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7 January 2021

Regulation of intestinal alpha-defensin activation by the metalloproteinase matrilysin in innate host defense

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7 January 2021

Heparan sulfate proteoglycans as extracellular docking molecules for matrilysin (matrix metalloproteinase 7)

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CD44 anchors the assembly of matrilysin/MMP-7 with heparin-binding epidermal growth factor precursor and ErbB4 and regulates female reproductive organ remodeling

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7 January 2021

Gene expression analysis reveals matrilysin as a key regulator of pulmonary fibrosis in mice and humans

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7 January 2021

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Identifiers

DOI

1 reference

17 May 2018

PubMed ID

12464176

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17 May 2018