IDDM2/insulin VNTR modifies risk conferred by IDDM1/HLA for development of Type 1 diabetes and associated autoimmunity. (Q53798911)

19 May 2018

title

IDDM2/insulin VNTR modifies risk conferred by IDDM1/HLA for development of Type 1 diabetes and associated autoimmunity. (English)

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19 May 2018

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7

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Ezio Bonifacio

10

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author name string

M Walter

1

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19 May 2018

E Albert

2

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M Conrad

3

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E Keller

4

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M Hummel

5

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K Ferber

6

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J A Todd

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A-G Ziegler

9

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publication date

16 May 2003

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19 May 2018

published in

Diabetologia

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19 May 2018

volume

46

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19 May 2018

issue

5

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19 May 2018

page(s)

712-720

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19 May 2018

https://scigraph.springernature.com/pub.10.1007/s00125-003-1082-z

exact match

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cites work

Seven regions of the genome show evidence of linkage to type 1 diabetes in a consensus analysis of 767 multiplex families

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Genetic control of autoimmunity in Type I diabetes and associated disorders

21 January 2018

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Early expression of antiinsulin autoantibodies of humans and the NOD mouse: evidence for early determination of subsequent diabetes

21 January 2018

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Genetic risk determines the emergence of diabetes-associated autoantibodies in young children

21 January 2018

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Analysis of HLA-DQ genotypes and susceptibility in insulin-dependent diabetes mellitus

21 January 2018

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A correlation between the relative predisposition of MHC class II alleles to type 1 diabetes and the structure of their proteins.

21 January 2018

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The aetiology of Type I diabetes

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The insulin gene is transcribed in the human thymus and transcription levels correlated with allelic variation at the INS VNTR-IDDM2 susceptibility locus for type 1 diabetes.

21 January 2018

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https://api.crossref.org/works/10.1007%2FS00125-003-1082-Z

Insulin expression in human thymus is modulated by INS VNTR alleles at the IDDM2 locus.

21 January 2018

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https://api.crossref.org/works/10.1007%2FS00125-003-1082-Z

Susceptibility to human type 1 diabetes at IDDM2 is determined by tandem repeat variation at the insulin gene minisatellite locus

21 January 2018

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Autoantibody appearance and risk for development of childhood diabetes in offspring of parents with type 1 diabetes: the 2-year analysis of the German BABYDIAB Study

21 January 2018

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Early expression and high prevalence of islet autoantibodies for DR3/4 heterozygous and DR4/4 homozygous offspring of parents with Type I diabetes: the German BABYDIAB study

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Conditional linkage disequilibrium analysis of a complex disease superlocus, IDDM1 in the HLA region, reveals the presence of independent modifying gene effects influencing the type 1 diabetes risk encoded by the major HLA-DQB1, -DRB1 disease loci

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12 December 2020

A Polymorphic Locus Near the Human Insulin Gene Is Associated with Insulin-dependent Diabetes Melliitus

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12 December 2020

Prevalence, characteristics and diabetes risk associated with transient maternally acquired islet antibodies and persistent islet antibodies in offspring of parents with type 1 diabetes

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Transient antiislet autoantibodies: infrequent occurrence and lack of association with "genetic" risk factors

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Two-locus maximum lod score analysis of a multifactorial trait: joint consideration of IDDM2 and IDDM4 with IDDM1 in type 1 diabetes.

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Genetic effects on age-dependent onset and islet cell autoantibody markers in type 1 diabetes

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Association of particular HLA class II alleles, haplotypes and genotypes with susceptibility to IDDM in the Belgian population

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Refining genetic analysis of type I diabetes

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Evidence for association between the class I subset of the insulin gene minisatellite (IDDM2 locus) and IDDM in the Japanese population

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Islet autoimmunity in infants with a Type I diabetic relative is common but is frequently restricted to one autoantibody

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10.1007/S00125-003-1082-Z

Identifiers

DOI

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19 May 2018

PubMed ID

12750767

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19 May 2018