Asymmetric dimethylarginine (ADMA) elevation and arginase up-regulation contribute to endothelial dysfunction related to insulin resistance in rats and morbidly obese humans. (Q53798961)

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scientific article published on 4 March 2016

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English	Asymmetric dimethylarginine (ADMA) elevation and arginase up-regulation contribute to endothelial dysfunction related to insulin resistance in rats and morbidly obese humans.	scientific article published on 4 March 2016

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scholarly article

1 reference

Europe PubMed Central

19 May 2018

http://europepmc.org/abstract/MED/26840628

Asymmetric dimethylarginine (ADMA) elevation and arginase up-regulation contribute to endothelial dysfunction related to insulin resistance in rats and morbidly obese humans. (English)

1 reference

Europe PubMed Central

19 May 2018

http://europepmc.org/abstract/MED/26840628

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insulin resistance

1 reference

based on heuristic

inferred from title

Leocadio Rodríguez-Mañas

8

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12 June 2020

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Mariam El Assar

1

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12 June 2020

Javier Angulo

2

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12 June 2020

Marta Santos-Ruiz

3

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12 June 2020

Juan Carlos Ruiz de Adana

4

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12 June 2020

María Luz Pindado

5

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12 June 2020

Alberto Sánchez-Ferrer

6

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12 June 2020

Alberto Hernández

7

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12 June 2020

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publication date

4 March 2016

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http://europepmc.org/abstract/MED/26840628

Journal of Physiology

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12 June 2020

594

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12 June 2020

3045-3060

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12 June 2020

11

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12 June 2020

Differential effect of amylin on endothelial-dependent vasodilation in mesenteric arteries from control and insulin resistant rats

1 reference

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FM19G11 reverses endothelial dysfunction in rat and human arteries through stimulation of the PI3K/Akt/eNOS pathway, independently of mTOR/HIF-1α activation.

1 reference

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Arginase inhibition restores endothelial function in diet-induced obesity

1 reference

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p38 mitogen-activated protein kinase is involved in arginase-II-mediated eNOS-uncoupling in obesity

1 reference

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Preserved endothelial function in human obesity in the absence of insulin resistance

1 reference

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Asymmetric dimethylarginine plasma levels and endothelial function in newly diagnosed type 2 diabetic patients

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Arginase II Promotes Macrophage Inflammatory Responses Through Mitochondrial Reactive Oxygen Species, Contributing to Insulin Resistance and Atherogenesis

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Positive crosstalk between arginase-II and S6K1 in vascular endothelial inflammation and aging

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In vivo nitric oxide synthesis, insulin sensitivity, and asymmetric dimethylarginine in obese subjects without and with metabolic syndrome.

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Weight loss improves endothelial function independently of ADMA reduction in severe obesity.

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Asymmetric dimethylarginine as a mediator of vascular dysfunction and a marker of cardiovascular disease and mortality: an intriguing interaction with diabetes mellitus

1 reference

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Vascular endothelial-specific dimethylarginine dimethylaminohydrolase-1-deficient mice reveal that vascular endothelium plays an important role in removing asymmetric dimethylarginine

1 reference

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Role of the PRMT-DDAH-ADMA axis in the regulation of endothelial nitric oxide production

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Diminished global arginine bioavailability and increased arginine catabolism as metabolic profile of increased cardiovascular risk

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Insulin reduces plasma arginase activity in type 2 diabetic patients

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The clinical significance of asymmetric dimethylarginine

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