p18INK4c and p27KIP1 are required for cell cycle arrest of differentiated myotubes. (Q53883186)

22 May 2018

title

p18INK4c and p27KIP1 are required for cell cycle arrest of differentiated myotubes. (English)

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22 May 2018

author name string

Terri K Myers

series ordinal

1

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22 May 2018

Sébastien E Andreuzza

series ordinal

2

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22 May 2018

David S Franklin

series ordinal

3

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22 May 2018

language of work or name

English

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publication date

1 November 2004

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22 May 2018

Experimental Cell Research

published in

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22 May 2018

volume

300

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22 May 2018

issue

2

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22 May 2018

page(s)

365-378

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22 May 2018

Skeletal muscle formation in vertebrates

cites work

1 reference

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Inhibition of myogenic differentiation in proliferating myoblasts by cyclin D1-dependent kinase

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Induction of p18INK4c and its predominant association with CDK4 and CDK6 during myogenic differentiation

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p53-independent expression of p21Cip1 in muscle and other terminally differentiating cells

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Correlation of terminal cell cycle arrest of skeletal muscle with induction of p21 by MyoD.

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Involvement of the cell-cycle inhibitor Cip1/WAF1 and the E1A-associated p300 protein in terminal differentiation.

7 January 2021

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Altered cell differentiation and proliferation in mice lacking p57KIP2 indicates a role in Beckwith–Wiedemann syndrome

7 January 2021

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Skeletal muscle cells lacking the retinoblastoma protein display defects in muscle gene expression and accumulate in S and G2 phases of the cell cycle

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SV40 immortalizes myogenic cells: DNA synthesis and mitosis in differentiating myotubes

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SV40 large T antigen reinduces the cell cycle in terminally differentiated myotubes through inducing Cdk2, Cdc2, and their partner cyclins

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Mitotic cycle reactivation in terminally differentiated cells by adenovirus infection

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Expression of E1A in terminally differentiated muscle cells reactivates the cell cycle and suppresses tissue-specific genes by separable mechanisms

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p21 and retinoblastoma protein control the absence of DNA replication in terminally differentiated muscle cells

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Myogenin expression, cell cycle withdrawal, and phenotypic differentiation are temporally separable events that precede cell fusion upon myogenesis.

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Mice lacking p27(Kip1) display increased body size, multiple organ hyperplasia, retinal dysplasia, and pituitary tumors

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A syndrome of multiorgan hyperplasia with features of gigantism, tumorigenesis, and female sterility in p27(Kip1)-deficient mice

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CDK inhibitors p18(INK4c) and p27(Kip1) mediate two separate pathways to collaboratively suppress pituitary tumorigenesis

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p21(CIP1) and p57(KIP2) control muscle differentiation at the myogenin step

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Resistance to apoptosis conferred by Cdk inhibitors during myocyte differentiation.

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Mice Lacking p21CIP1/WAF1 undergo normal development, but are defective in G1 checkpoint control

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Myogenesis: fusion, myosin synthesis, and the mitotic cycle

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Reconstitution of cyclin D1-associated kinase activity drives terminally differentiated cells into the cell cycle

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10.1016/J.YEXCR.2004.07.024

7 January 2021

Identifiers

DOI

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22 May 2018

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22 May 2018