Bradykinin pretreatment improves ischemia tolerance of the rabbit heart by tyrosine kinase mediated pathways. (Q53921208)

22 May 2018

title

Bradykinin pretreatment improves ischemia tolerance of the rabbit heart by tyrosine kinase mediated pathways. (English)

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22 May 2018

author

Jun Feng

series ordinal

1

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22 May 2018

author name string

Rosenkranz ER

series ordinal

2

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22 May 2018

language of work or name

English

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publication date

1 November 1999

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22 May 2018

The Annals of Thoracic Surgery

published in

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22 May 2018

volume

68

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22 May 2018

issue

5

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22 May 2018

page(s)

1567-1572

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22 May 2018

Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium

cites work

1 reference

https://api.crossref.org/works/10.1016%2FS0003-4975%2899%2901041-3

Evidence that translocation of protein kinase C is a key event during ischemic preconditioning of rabbit myocardium

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0003-4975%2899%2901041-3

Ischemic Preconditioning Induces Selective Translocation of Protein Kinase C Isoforms and in the Heart of Conscious Rabbits Without Subcellular Redistribution of Total Protein Kinase C Activity

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0003-4975%2899%2901041-3

Prevention of ischemic preconditioning only by combined inhibition of protein kinase C and protein tyrosine kinase in pigs

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0003-4975%2899%2901041-3

Protein tyrosine kinase is downstream of protein kinase C for ischemic preconditioning's anti-infarct effect in the rabbit heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0003-4975%2899%2901041-3

The role of protein kinase C in ischemic preconditioning

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0003-4975%2899%2901041-3

Bradykinin metabolism pathway in the rat pulmonary circulation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0003-4975%2899%2901041-3

Reduction of infarct size by local angiotensin-converting enzyme inhibition is abolished by a bradykinin antagonist

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0003-4975%2899%2901041-3

Bradykinin-mediated cardiovascular protective actions of ACE inhibitors. A new dimension in anti-ischaemic therapy?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0003-4975%2899%2901041-3

Role of kinins in the pathophysiology of myocardial ischemia. In vitro and in vivo studies

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0003-4975%2899%2901041-3

Tyrosine Phosphorylation and Bradykinin-Induced Signaling in Endothelial Cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0003-4975%2899%2901041-3

Protection against infarction afforded by preconditioning is mediated by A1 adenosine receptors in rabbit heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0003-4975%2899%2901041-3

Role of adenosine receptor activation in myocardial infarct size limitation by ischaemic preconditioning

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0003-4975%2899%2901041-3

Ischemic preconditioning triggers the activation of MAP kinases and MAPKAP kinase 2 in rat hearts

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0003-4975%2899%2901041-3

PDGF stimulation of inositol phospholipid hydrolysis requires PLC-gamma 1 phosphorylation on tyrosine residues 783 and 1254.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0003-4975%2899%2901041-3

Phosphorylation of tyrosine 182 of p38 mitogen-activated protein kinase correlates with the protection of preconditioning in the rabbit heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0003-4975%2899%2901041-3

Signal transduction in ischemic preconditioning.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0003-4975%2899%2901041-3

Are the inotropic and antiarrhythmic effects of bradykinin due to increases in coronary flow?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0003-4975%2899%2901041-3

10.1016/S0003-4975(99)01041-3

7 January 2021

Identifiers

DOI

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22 May 2018

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22 May 2018