Involvement of c-Fos in signaling grp78 induction following ER calcium release. (Q54025967)

23 May 2018

title

Involvement of c-Fos in signaling grp78 induction following ER calcium release. (English)

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23 May 2018

author name string

He H

series ordinal

1

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23 May 2018

McColl K

series ordinal

2

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23 May 2018

Distelhorst CW

series ordinal

3

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23 May 2018

language of work or name

English

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publication date

1 November 2000

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23 May 2018

published in

Oncogene

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23 May 2018

volume

19

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23 May 2018

issue

51

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23 May 2018

page(s)

5936-5943

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23 May 2018

https://scigraph.springernature.com/pub.10.1038/sj.onc.1203994

exact match

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cites work

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Transactivation of the grp78 promoter by malfolded proteins, glycosylation block, and calcium ionophore is mediated through a proximal region containing a CCAAT motif which interacts with CTF/NF-I

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The endoplasmic reticulum stress-responsive protein GRP78 protects neurons against excitotoxicity and apoptosis: suppression of oxidative stress and stabilization of calcium homeostasis.

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Interaction of ATF6 and serum response factor

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Cascade induction of c-fos, c-myc, and heat shock 70K transcripts during regression of the rat ventral prostate gland

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c-Fos degradation by the proteasome. An early, Bcl-2-regulated step in apoptosis

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Effect of glucocorticosteroid treatment on intracellular calcium homeostasis in mouse lymphoma cells

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BiP, a major chaperone protein of the endoplasmic reticulum lumen, plays a direct and important role in the storage of the rapidly exchanging pool of Ca2+

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Mouse Lymphoma Cells Destined to Undergo Apoptosis in Response to Thapsigargin Treatment Fail to Generate a Calcium-mediatedgrp78/grp94Stress Response

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Immunoglobulin binding protein (BiP) function is required to protect cells from endoplasmic reticulum stress but is not required for the secretion of selective proteins

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Signal transduction from the endoplasmic reticulum to the cell nucleus

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Bcl-2 inhibits c-fos induction by calcium

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Rapid attenuation of AP-1 transcriptional factors associated with nitric oxide (NO)-mediated neuronal cell death

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Identifiers

DOI

10.1038/SJ.ONC.1203994

1 reference

23 May 2018

PubMed ID

11127825

1 reference

23 May 2018