MiR-26a inhibits prostate cancer progression by repression of Wnt5a. (Q54200488)

26 May 2018

title

MiR-26a inhibits prostate cancer progression by repression of Wnt5a. (English)

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26 May 2018

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7

Li Li

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26 May 2018

Shijia Zhao

1

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26 May 2018

Xiangdong Ye

2

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26 May 2018

Lei Xiao

3

1 reference

26 May 2018

Xuexiong Lian

4

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26 May 2018

Yupeng Feng

5

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26 May 2018

Feng Li

6

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26 May 2018

publication date

28 June 2014

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26 May 2018

published in

Tumor Biology

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26 May 2018

volume

35

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26 May 2018

issue

10

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26 May 2018

page(s)

9725-9733

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26 May 2018

https://scigraph.springernature.com/pub.10.1007/s13277-014-2206-4

exact match

0 references

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Ectopic expression of miR-126*, an intronic product of the vascular endothelial EGF-like 7 gene, regulates prostein translation and invasiveness of prostate cancer LNCaP cells

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MicroRNA-21 promotes cell transformation by targeting the programmed cell death 4 gene

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MicroRNA-21 directly targets MARCKS and promotes apoptosis resistance and invasion in prostate cancer cells

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Widespread deregulation of microRNA expression in human prostate cancer.

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Genomic loss of microRNA-101 leads to overexpression of histone methyltransferase EZH2 in cancer

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MicroRNA in ischemic stroke etiology and pathology

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Expression profile analysis reveals putative prostate cancer-related microRNAs

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Circulating microRNAs (miRNA) in serum of patients with prostate cancer

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Elevated expression of prostate cancer-associated genes is linked to down-regulation of microRNAs

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Overexpression of MYC and EZH2 cooperates to epigenetically silence MST1 expression

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Genome-wide DNA methylation events in TMPRSS2-ERG fusion-negative prostate cancers implicate an EZH2-dependent mechanism with miR-26a hypermethylation.

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Myc enforces overexpression of EZH2 in early prostatic neoplasia via transcriptional and post-transcriptional mechanisms

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miR-26a enhances miRNA biogenesis by targeting Lin28B and Zcchc11 to suppress tumor growth and metastasis.

21 January 2018

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Four microRNAs promote prostate cell proliferation with regulation of PTEN and its downstream signals in vitro

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21 January 2018

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PubMed

https://pubmed.ncbi.nlm.nih.gov/24972966

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Cellular transitions and tissue engineering

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/24972966

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Molecular biology of prostate cancer progression.

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/24972966

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1007/S13277-014-2206-4

1 reference

26 May 2018

PubMed ID

24972966

1 reference

26 May 2018