Oxygen tension controls the expression of the monocarboxylate transporter MCT4 in cultured mouse cortical astrocytes via a hypoxia-inducible factor-1α-mediated transcriptional regulation. (Q54388530)

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English	Oxygen tension controls the expression of the monocarboxylate transporter MCT4 in cultured mouse cortical astrocytes via a hypoxia-inducible factor-1α-mediated transcriptional regulation.	scientific article

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scholarly article

1 reference

Europe PubMed Central

29 May 2018

http://europepmc.org/abstract/MED/24375723

Oxygen tension controls the expression of the monocarboxylate transporter MCT4 in cultured mouse cortical astrocytes via a hypoxia-inducible factor-1α-mediated transcriptional regulation. (English)

1 reference

Europe PubMed Central

29 May 2018

http://europepmc.org/abstract/MED/24375723

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Europe PubMed Central

29 May 2018

http://europepmc.org/abstract/MED/24375723

author name string

Katia Rosafio

1

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29 May 2018

http://europepmc.org/abstract/MED/24375723

publication date

21 December 2013

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Europe PubMed Central

29 May 2018

http://europepmc.org/abstract/MED/24375723

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Europe PubMed Central

29 May 2018

http://europepmc.org/abstract/MED/24375723

62

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Europe PubMed Central

29 May 2018

http://europepmc.org/abstract/MED/24375723

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Europe PubMed Central

29 May 2018

http://europepmc.org/abstract/MED/24375723

477-490

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Europe PubMed Central

29 May 2018

http://europepmc.org/abstract/MED/24375723

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Glucose transporter 1 and monocarboxylate transporters 1, 2, and 4 localization within the glial cells of shark blood-brain-barriers.

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Endothelial cell-derived nitric oxide enhances aerobic glycolysis in astrocytes via HIF-1α-mediated target gene activation.

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Hypoxic injury to developing glial cells: protective effect of high glucose.

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Why is the partial oxygen pressure of human tissues a crucial parameter? Small molecules and hypoxia

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Tumor hypoxia and metabolism -- towards novel anticancer approaches

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The low-affinity monocarboxylate transporter MCT4 is adapted to the export of lactate in highly glycolytic cells

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The effects of extracellular acidosis on neurons and glia in vitro

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The SLC16 gene family?from monocarboxylate transporters (MCTs) to aromatic amino acid transporters and beyond

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Dichloroacetate effects on glucose and lactate oxidation by neurons and astroglia in vitro and on glucose utilization by brain in vivo

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CD147 subunit of lactate/H+ symporters MCT1 and hypoxia-inducible MCT4 is critical for energetics and growth of glycolytic tumors

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AMP-activated protein kinase activity is critical for hypoxia-inducible factor-1 transcriptional activity and its target gene expression under hypoxic conditions in DU145 cells

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Nitric oxide induces the expression of the monocarboxylate transporter MCT4 in cultured astrocytes by a cGMP-independent transcriptional activation

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Changes in MCT 1, MCT 4, and LDH expression are tissue specific in rats after long-term hypobaric hypoxia

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Brain tissue oxygen concentration measurements.

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AMP-dependent protein kinase alpha 2 isoform promotes hypoxia-induced VEGF expression in human glioblastoma

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Tissue-specific transcriptional regulation of monocarboxylate transporters (MCTs) during short-term hypoxia in zebrafish (Danio rerio).

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Cellular and subcellular distribution of monocarboxylate transporters in cultured brain cells and in the adult brain.

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Glutamate uptake into astrocytes stimulates aerobic glycolysis: a mechanism coupling neuronal activity to glucose utilization

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Hypoxia stimulates lactate release and modulates monocarboxylate transporter (MCT1, MCT2, and MCT4) expression in human adipocytes

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Role of hypoxia-induced anorexia and right ventricular hypertrophy on lactate transport and MCT expression in rat muscle.

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Highly differential expression of the monocarboxylate transporters MCT2 and MCT4 in the developing rat brain

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Differential effects of iodoacetamide and iodoacetate on glycolysis and glutathione metabolism of cultured astrocytes.

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Glia are the main source of lactate utilized by neurons for recovery of function posthypoxia.

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Preischemic hyperglycemia-aggravated damage: evidence that lactate utilization is beneficial and glucose-induced corticosterone release is detrimental

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Regulation of oxygen homeostasis by hypoxia-inducible factor 1.

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Characterization of the glycogenolysis elicited by vasoactive intestinal peptide, noradrenaline and adenosine in primary cultures of mouse cerebral cortical astrocytes.

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Acute feedback control of astrocytic glycolysis by lactate.

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The plasma membrane lactate transporter MCT4, but not MCT1, is up-regulated by hypoxia through a HIF-1alpha-dependent mechanism.

1 reference

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21 January 2018

Differential metabolic adaptation to acute and long-term hypoxia in rat primary cortical astrocytes.

1 reference

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Simulation of aspects of ischemia in cell culture: changes in lactate compartmentation.

1 reference

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Lactic acid efflux from white skeletal muscle is catalyzed by the monocarboxylate transporter isoform MCT3.

1 reference

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Arachidonic acid stimulates glucose uptake in cerebral cortical astrocytes

1 reference

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Cell-specific localization of monocarboxylate transporters, MCT1 and MCT2, in the adult mouse brain revealed by double immunohistochemical labeling and confocal microscopy

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

Acidosis causes failure of astrocyte glutamate uptake during hypoxia

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

Identifiers

10.1002/GLIA.22618

1 reference

Europe PubMed Central

29 May 2018

http://europepmc.org/abstract/MED/24375723

1 reference

Europe PubMed Central

29 May 2018

http://europepmc.org/abstract/MED/24375723

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