Shear stress-mediated F1/FO ATP synthase-dependent CO2 gas excretion from human pulmonary arteriolar endothelial cells. (Q54573253)

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title

Shear stress-mediated F1/FO ATP synthase-dependent CO2 gas excretion from human pulmonary arteriolar endothelial cells. (English)

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author name string

Yoshiko Kawai

1

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Kazuo Yoshida

2

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Maki Kaidoh

3

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Yumiko Yokoyama

4

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Toshio Ohhashi

5

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publication date

1 May 2012

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Journal of Cellular Physiology

published in

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volume

227

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issue

5

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page(s)

2059-2068

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A fusion protein of the human P2Y(1) receptor and NTPDase1 exhibits functional activities of the native receptor and ectoenzyme and reduced signaling responses to endogenously released nucleotides

cites work

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Direct evidence of participation of rat lung carbonic anhydrase in CO2 reactions

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Isolation of murine lung endothelial cells

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MDA-MB-231 produces ATP-mediated ICAM-1-dependent facilitation of the attachment of carcinoma cells to human lymphatic endothelial cells

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Shear stress-induced ATP-mediated endothelial constitutive nitric oxide synthase expression in human lymphatic endothelial cells

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Metabolic control of excessive extracellular nucleotide accumulation by CD39/ecto-nucleotidase-1: implications for ischemic vascular diseases

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Lung endothelial cell proliferation with decreased shear stress is mediated by reactive oxygen species

21 January 2018

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Endothelial cell surface F1-F0 ATP synthase is active in ATP synthesis and is inhibited by angiostatin

21 January 2018

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Genetic evidence supporting caveolae microdomain regulation of calcium entry in endothelial cells

21 January 2018

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Cloning, sequencing, and expression of a human brain ecto-apyrase related to both the ecto-ATPases and CD39 ecto-apyrases1.

21 January 2018

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Involvement of cell surface ATP synthase in flow-induced ATP release by vascular endothelial cells.

21 January 2018

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21 January 2018

Identifiers

DOI

10.1002/JCP.22937

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