Transgenic mice with overexpression of mutated human optineurin(E50K) in the retina. (Q54580795)

1 June 2018

title

Transgenic mice with overexpression of mutated human optineurin(E50K) in the retina. (English)

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1 June 2018

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Qingfeng Meng

1

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1 June 2018

Zheng Xiao

2

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1 June 2018

Huiping Yuan

3

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1 June 2018

Fei Xue

4

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1 June 2018

Yuanmao Zhu

5

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1 June 2018

Xinrong Zhou

6

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1 June 2018

Binbin Yang

7

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1 June 2018

Jingbo Sun

8

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1 June 2018

Bo Meng

9

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1 June 2018

Xian Sun

10

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Fang Cheng

11

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1 June 2018

publication date

17 June 2011

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1 June 2018

Molecular Biology Reports

published in

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1 June 2018

volume

39

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1 June 2018

issue

2

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1 June 2018

page(s)

1119-1124

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1 June 2018

The Ocular Hypertension Treatment Study: baseline factors that predict the onset of primary open-angle glaucoma.

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Adult-onset primary open-angle glaucoma caused by mutations in optineurin

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Optineurin links myosin VI to the Golgi complex and is involved in Golgi organization and exocytosis

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Optineurin negatively regulates TNFalpha- induced NF-kappaB activation by competing with NEMO for ubiquitinated RIP

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Studies of optineurin, a glaucoma gene: Golgi fragmentation and cell death from overexpression of wild-type and mutant optineurin in two ocular cell types

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Molecular genetic analysis of optineurin gene for primary open-angle and normal tension glaucoma in the Japanese population

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inferred from PubMed ID database lookup

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Global data on blindness

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Transcription of retinoic acid receptor genes in transgenic mice increases CD8 T-cell subset

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inferred from PubMed ID database lookup

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based on heuristic

Identifiers

DOI

10.1007/S11033-011-0840-0

1 reference

1 June 2018

1 reference

1 June 2018