Role of MTHFR polymorphisms and folate levels in different phenotypes of sporadic colorectal cancers. (Q54585070)

1 June 2018

title

Role of MTHFR polymorphisms and folate levels in different phenotypes of sporadic colorectal cancers. (English)

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1 June 2018

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Shih-Ching Chang

1

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1 June 2018

Pei-Ching Lin

2

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Jen-Kou Lin

3

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Shung-Haur Yang

4

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Huann-Sheng Wang

5

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Anna Fen-Yau Li

6

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publication date

29 August 2006

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International Journal of Colorectal Disease

published in

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volume

22

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issue

5

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page(s)

483-489

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https://scigraph.springernature.com/pub.10.1007/s00384-006-0190-x

exact match

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Aneuploidy vs. gene mutation hypothesis of cancer: recent study claims mutation but is found to support aneuploidy

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A polymorphism of the methionine synthase gene: association with plasma folate, vitamin B12, homocyst(e)ine, and colorectal cancer risk

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Folate deficiency causes uracil misincorporation into human DNA and chromosome breakage: implications for cancer and neuronal damage

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Associations of plasma homocysteine and the methylenetetrahydrofolate reductase C677T polymorphism with carotid intima media thickness among South Asian, Chinese and European Canadians

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Relationship between genetic alterations and prognosis in sporadic colorectal cancer.

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Plasma homocysteine is not subject to seasonal variation

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Dietary monoglutamate and polyglutamate folate are associated with plasma folate concentrations in Dutch men and women aged 20-65 years

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BRAF mutation is frequently present in sporadic colorectal cancer with methylated hMLH1, but not in hereditary nonpolyposis colorectal cancer

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Low intake of calcium, folate, nicotinic acid, vitamin E, retinol, beta-carotene and high intake of pantothenic acid, biotin and riboflavin are significantly associated with increased genome instability--results from a dietary intake and micronucleu

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A three-day weighed food record and a semiquantitative food-frequency questionnaire are valid measures for assessing the folate and vitamin B-12 intakes of women aged 16 to 19 years

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Folate deficiency enhances the development of colonic neoplasia in dimethylhydrazine-treated rats

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Methylation of the hMLH1 promoter correlates with lack of expression of hMLH1 in sporadic colon tumors and mismatch repair-defective human tumor cell lines

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Mutations of BRAF are associated with extensive hMLH1 promoter methylation in sporadic colorectal carcinomas.

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Folate status: effects on pathways of colorectal carcinogenesis

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Influence of a methionine synthase (D919G) polymorphism on plasma homocysteine and folate levels and relation to risk of myocardial infarction

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5,10-methylenetetrahydrofolate reductase 677 and 1298 polymorphisms, folate intake, and microsatellite instability in colon cancer

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Methylenetetrahydrofolate reductase polymorphism (MTHFR C677T) and bone mineral density in Chinese men and women

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Lessons from hereditary colorectal cancer

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The C677T mutation of the methylenetetrahydrofolate reductase gene is not associated with the risk of coronary artery disease or venous thrombosis among Chinese in Taiwan

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Genetic instability in colorectal cancers

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Nutrition classics. Transactions of the Association of American Physicians, volume 75, 1962, pages 307-320. Experimental nutritional folate deficiency in man. by Victor Herbert

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Dietary folate protects against the development of macroscopic colonic neoplasia in a dose responsive manner in rats

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Abnormal folate metabolism and mutation in the methylenetetrahydrofolate reductase gene may be maternal risk factors for Down syndrome

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Genomic instability and alterations in Apc, Mcc and Dcc in Hong Kong patients with colorectal carcinoma.

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Folate, DNA stability and colo-rectal neoplasia

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Methylenetetrahydrofolate reductase, diet, and risk of colon cancer

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Genomic changes defining the genesis, progression, and malignancy potential in solid human tumors: a phenotype/genotype correlation

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A polymorphism in the methylenetetrahydrofolate reductase gene predisposes to colorectal cancers with microsatellite instability

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Folate, vitamin B12 and vitamin B6 and one carbon metabolism

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12 December 2020

10.1007/S00384-006-0190-X

Identifiers

DOI

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1 June 2018

PubMed ID

16941173

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1 June 2018