Evidence of NK cell dysfunction in SIV-infected rhesus monkeys: impairment of cytokine secretion and NKG2C/C2 expression. (Q54587667)

1 June 2018

title

Evidence of NK cell dysfunction in SIV-infected rhesus monkeys: impairment of cytokine secretion and NKG2C/C2 expression. (English)

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1 June 2018

author

Paul F McKay

series ordinal

2

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1 June 2018

Norman L. Letvin

series ordinal

3

object named as

Norman L Letvin

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1 June 2018

author name string

Michelle L LaBonte

series ordinal

1

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1 June 2018

language of work or name

English

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publication date

1 September 2006

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1 June 2018

European Journal of Immunology

published in

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1 June 2018

volume

36

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1 June 2018

page(s)

2424-2433

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1 June 2018

issue

9

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1 June 2018

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The impaired NK cell cytolytic function in viremic HIV-1 infection is associated with a reduced surface expression of natural cytotoxicity receptors (NKp46, NKp30 and NKp44)

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Characterization of rhesus monkey CD94/NKG2 family members and identification of novel transmembrane-deleted forms of NKG2-A, B, C, and D.

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Selective expression of NKG2-A and NKG2-C mRNAs and novel alternative splicing of 5' exons in rhesus monkey decidua

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Anatomy of a murder--signal transduction pathways leading to activation of natural killer cells.

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Mitogen-activated protein kinase activity is involved in effector functions triggered by the CD94/NKG2-C NK receptor specific for HLA-E.

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The selective downregulation of class I major histocompatibility complex proteins by HIV-1 protects HIV-infected cells from NK cells

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Killing of human immunodeficiency virus-infected primary T-cell blasts by autologous natural killer cells is dependent on the ability of the virus to alter the expression of major histocompatibility complex class I molecules

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HLA-C and HLA-E reduce antibody-dependent natural killer cell-mediated cytotoxicity of HIV-infected primary T cell blasts

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HLA-E Up-Regulation Induced by HIV Infection May Directly Contribute to CD94-Mediated Impairment of NK Cells

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Identifiers

DOI

10.1002/EJI.200635901

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1 June 2018

PubMed ID

16906533

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1 June 2018