Molecular mechanisms of activation of endothelial nitric oxide synthase mediated by transient receptor potential vanilloid type 1. (Q54596498)

1 June 2018

title

Molecular mechanisms of activation of endothelial nitric oxide synthase mediated by transient receptor potential vanilloid type 1. (English)

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author name string

Li-Chieh Ching

1

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Song-Kun Shyue

3

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Kuo-Hui Su

4

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Jeng Wei

5

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Li-Ching Cheng

6

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Yuan-Bin Yu

7

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Ching-Chian Pan

8

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Tzong-Shyuan Lee

9

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publication date

14 April 2011

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published in

Cardiovascular Research

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volume

91

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page(s)

492-501

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issue

3

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Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factor

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Valsartan regulates the interaction of angiotensin II type 1 receptor and endothelial nitric oxide synthase via Src/PI3K/Akt signalling

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Phosphorylation of Thr(495) regulates Ca(2+)/calmodulin-dependent endothelial nitric oxide synthase activity.

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Domain mapping studies reveal that the M domain of hsp90 serves as a molecular scaffold to regulate Akt-dependent phosphorylation of endothelial nitric oxide synthase and NO release

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Functional interaction of endothelial nitric oxide synthase with a voltage-dependent anion channel.

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Evodiamine functions as an agonist for the vanilloid receptor TRPV1.

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Ca2+/calmodulin modulates TRPV1 activation by capsaicin.

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Phosphoinositide 3-kinase binds to TRPV1 and mediates NGF-stimulated TRPV1 trafficking to the plasma membrane.

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Phosphorylation of vanilloid receptor 1 by Ca2+/calmodulin-dependent kinase II regulates its vanilloid binding

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Anandamide elicits an acute release of nitric oxide through endothelial TRPV1 receptor activation in the rat arterial mesenteric bed

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TRP channel and cardiovascular disease

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Nitric oxide activates TRP channels by cysteine S-nitrosylation.

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Transient receptor potential vanilloid type 4-deficient mice exhibit impaired endothelium-dependent relaxation induced by acetylcholine in vitro and in vivo

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Endothelium-dependent cerebral artery dilation mediated by TRPA1 and Ca2+-Activated K+ channels

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Molecular mechanisms involved in the regulation of the endothelial nitric oxide synthase

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CaM Kinase II-dependent pathophysiological signalling in endothelial cells

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Inhibitory interactions of the bradykinin B2 receptor with endothelial nitric-oxide synthase

21 January 2018

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A novel mechanism contributing to development of Dahl salt-sensitive hypertension: role of the transient receptor potential vanilloid type 1.

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Transient receptor potential vanilloid gene deletion exacerbates inflammation and atypical cardiac remodeling after myocardial infarction

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Activation of TRPV1 by dietary capsaicin improves endothelium-dependent vasorelaxation and prevents hypertension.

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21 January 2018

Identifiers

DOI

10.1093/CVR/CVR104

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1 June 2018

PubMed ID

21493704

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1 June 2018