Role of tumour necrosis factor and reactive oxygen intermediates in lipopolysaccharide-induced pulmonary oedema and lethality. (Q54661310)

2 June 2018

title

Role of tumour necrosis factor and reactive oxygen intermediates in lipopolysaccharide-induced pulmonary oedema and lethality. (English)

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2 June 2018

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4

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author name string

Gatti S

series ordinal

1

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2 June 2018

Faggioni R

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2

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Echtenacher B

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3

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publication date

1 March 1993

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2 June 2018

Clinical and Experimental Immunology

published in

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2 June 2018

volume

91

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2 June 2018

issue

3

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2 June 2018

page(s)

456-461

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2 June 2018

Plasma tumor necrosis factor in patients with septic shock. Mortality rate, incidence of adult respiratory distress syndrome, and effects of methylprednisolone administration.

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Tumor necrosis factor causes increased pulmonary permeability and edema. Comparison to septic acute lung injury

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Increased sheep lung vascular permeability caused by Escherichia coli endotoxin

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Cyclophosphamide-induced lung damage in mice: protection by a small preliminary dose

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The toxic effects of tumor necrosis factor in vivo and their prevention by cyclooxygenase inhibitors

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Antioxidants protect cultured bovine lung endothelial cells from injury by endotoxin

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Synergy of interleukin 1 and granulocyte colony-stimulating factor: in vivo stimulation of stem-cell recovery and hematopoietic regeneration following 5-fluorouracil treatment of mice

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Adult respiratory distress syndrome in pediatric patients. I. Clinical aspects, pathophysiology, pathology, and mechanisms of lung injury

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A low dose of recombinant interleukin 1 protects granulocytopenic mice from lethal gram-negative infection

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Provocation of pulmonary vascular endothelial injury in rabbits by human recombinant interleukin-1 beta

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Recombinant tumor necrosis factor increases pulmonary vascular permeability independent of neutrophils

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The role of oxidant injury in tumor cell sensitivity to recombinant human tumor necrosis factor in vivo. Implications for mechanisms of action

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Effect of N-acetylcysteine on the pulmonary response to endotoxin in the awake sheep and upon in vitro granulocyte function

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Vascular hyperpermeability induced by tumor necrosis factor and its augmentation by IL-1 and IFN-gamma is inhibited by selective depletion of neutrophils with a monoclonal antibody

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Requirement of endogenous tumor necrosis factor/cachectin for recovery from experimental peritonitis

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Evidence for tumor necrosis factor-induced pulmonary microvascular injury after intestinal ischemia-reperfusion injury.

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Detection of interleukin 8 and tumor necrosis factor in normal humans after intravenous endotoxin: the effect of antiinflammatory agents

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Protective effect of chlorpromazine on endotoxin toxicity and TNF production in glucocorticoid-sensitive and glucocorticoid-resistant models of endotoxic shock

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Septic shock: treatment

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10.1111/J.1365-2249.1993.TB05924.X

Identifiers

DOI

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2 June 2018

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2 June 2018