Overexpression of VEGF-A in podocytes of adult mice causes glomerular disease. (Q54682241)

2 June 2018

title

Overexpression of VEGF-A in podocytes of adult mice causes glomerular disease. (English)

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2 June 2018

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8

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author name string

Delma Veron

1

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2 June 2018

Kimberly J Reidy

2

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2 June 2018

Claudia Bertuccio

3

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2 June 2018

Jason Teichman

4

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2 June 2018

Guillermo Villegas

5

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2 June 2018

Juan Jimenez

6

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2 June 2018

Wa Shen

7

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2 June 2018

David B Thomas

9

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2 June 2018

Alda Tufro

10

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2 June 2018

language of work or name

English

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publication date

10 March 2010

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2 June 2018

published in

Kidney International

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2 June 2018

volume

77

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2 June 2018

issue

11

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2 June 2018

page(s)

989-999

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2 June 2018

https://scigraph.springernature.com/pub.10.1038/ki.2010.64

exact match

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cites work

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Heterozygous embryonic lethality induced by targeted inactivation of the VEGF gene

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Glomerular-specific alterations of VEGF-A expression lead to distinct congenital and acquired renal diseases

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Toward a mouse model of diabetic nephropathy: is endothelial nitric oxide synthase the missing link?

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Increased renal expression of vascular endothelial growth factor (VEGF) and its receptor VEGFR-2 in experimental diabetes

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Amelioration of long-term renal changes in obese type 2 diabetic mice by a neutralizing vascular endothelial growth factor antibody

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Blockade of vascular endothelial growth factor signaling ameliorates diabetic albuminuria in mice

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VEGF inhibition and renal thrombotic microangiopathy

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Renal effects of anti-angiogenesis therapy: update for the internist

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Uncoupling of the VEGF-endothelial nitric oxide axis in diabetic nephropathy: an explanation for the paradoxical effects of VEGF in renal disease

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VEGF spatially directs angiogenesis during metanephric development in vitro

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Temporal and spatial regulation of VEGF-A controls vascular patterning in the embryonic lung

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Failure of blood-island formation and vasculogenesis in Flk-1-deficient mice

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Increased expression of vascular endothelial growth factor in kidney leads to progressive impairment of glomerular functions.

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Metalloproteinase inhibition ameliorates hypertension and prevents vascular dysfunction and remodeling in renovascular hypertensive rats.

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Podocyte-derived vascular endothelial growth factor mediates the stimulation of alpha3(IV) collagen production by transforming growth factor-beta1 in mouse podocytes

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The vascular endothelial growth factor VEGF165 induces perlecan synthesis via VEGF receptor-2 in cultured human brain microvascular endothelial cells

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Expression of podocyte-associated molecules in acquired human kidney diseases

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Nephrin ectodomain engagement results in Src kinase activation, nephrin phosphorylation, Nck recruitment, and actin polymerization

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Nck adaptor proteins link nephrin to the actin cytoskeleton of kidney podocytes

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Vascular permeability factor (VPF)/vascular endothelial growth factor (VEGF) peceptor-1 down-modulates VPF/VEGF receptor-2-mediated endothelial cell proliferation, but not migration, through phosphatidylinositol 3-kinase-dependent pathways

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VEGF receptor signalling - in control of vascular function

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Inducible overexpression of sFlt-1 in podocytes ameliorates glomerulopathy in diabetic mice

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Synergistic effect of angiotensin II and nitric oxide synthase inhibitor in increasing aortic stiffness in mice.

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An overview of real-time quantitative PCR: applications to quantify cytokine gene expression

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Identifiers

DOI

10.1038/KI.2010.64

1 reference

2 June 2018

release_6gxbxicxsrbl7dy6rf6kc5fix4

Fatcat ID

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/6gxbxicxsrbl7dy6rf6kc5fix4

24 November 2022

mapped directly with Wikidata item

PubMed ID

20375978

1 reference

2 June 2018