Molecular mechanisms underlying K+ current downregulation in canine tachycardia-induced heart failure. (Q54682988)

2 June 2018

title

Molecular mechanisms underlying K+ current downregulation in canine tachycardia-induced heart failure. (English)

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2 June 2018

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Fadi G Akar

1

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2 June 2018

Richard C Wu

2

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2 June 2018

George J Juang

3

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Yanli Tian

4

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Mirka Burysek

5

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Deborah Disilvestre

6

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Wei Xiong

7

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Antonis A Armoundas

8

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Gordon F Tomaselli

9

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publication date

28 January 2005

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2 June 2018

American Journal of Physiology Heart and Circulatory Physiology

published in

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volume

288

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page(s)

H2887-96

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issue

6

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MiRP1 forms IKr potassium channels with HERG and is associated with cardiac arrhythmia

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Unique topographical distribution of M cells underlies reentrant mechanism of torsade de pointes in the long-QT syndrome

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Electrical remodeling in hearts from a calcium-dependent mouse model of hypertrophy and failure: complex nature of K+ current changes and action potential duration

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A molecular basis for cardiac arrhythmia: HERG mutations cause long QT syndrome

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Regulation of Kv4.3 current by KChIP2 splice variants: a component of native cardiac I(to)?

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Modulation of Kv4.3 current by accessory subunits

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Alterations in cardiac gene expression during ventricular remodeling following experimental myocardial infarction

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Molecular basis of transient outward potassium current downregulation in human heart failure: a decrease in Kv4.3 mRNA correlates with a reduction in current density

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Ionic mechanism of action potential prolongation in ventricular myocytes from dogs with pacing-induced heart failure

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Characterization of inwardly rectifying K+ channel in human cardiac myocytes. Alterations in channel behavior in myocytes isolated from patients with idiopathic dilated cardiomyopathy.

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21 January 2018

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Canine ventricular KCNE2 expression resides predominantly in Purkinje fibers.

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Concordant expression of KChIP2 mRNA, protein and transient outward current throughout the canine ventricle.

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Regulation of KChIP2 potassium channel beta subunit gene expression underlies the gradient of transient outward current in canine and human ventricle

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Potential molecular basis of different physiological properties of the transient outward K+ current in rabbit and human atrial myocytes.

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Transmural expression of transient outward potassium current subunits in normal and failing canine and human hearts

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10.1152/AJPHEART.00320.2004

21 January 2018

Identifiers

DOI

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2 June 2018

PubMed ID

15681701

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2 June 2018