Lower serum zinc in major depression in relation to changes in serum acute phase proteins (Q56767348)

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4 November 2019

title

Lower serum zinc in major depression in relation to changes in serum acute phase proteins (English)

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4 November 2019

1

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4 November 2019

Hugo Neels

5

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4 November 2019

author name string

N De Vos

2

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4 November 2019

P Demedts

3

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4 November 2019

A Wauters

4

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4 November 2019

publication date

1 December 1999

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Journal of Affective Disorders

4 November 2019

published in

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4 November 2019

volume

56

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4 November 2019

issue

2-3

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4 November 2019

page(s)

189-194

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Immune markers in fibromyalgia: comparison with major depressed patients and normal volunteers.

4 November 2019

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Elevated levels of acute phase plasma proteins in major depression

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Regulation of the acute phase response by cytokines

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Altered zinc metabolism in mood disorder patients

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Interleukin-1 beta: a putative mediator of HPA axis hyperactivity in major depression?

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Hypozincemia in depression

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Lower serum zinc in major depression is a sensitive marker of treatment resistance and of the immune/inflammatory response in that illness.

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Pindolol and mianserin augment the antidepressant activity of fluoxetine in hospitalized major depressed patients, including those with treatment resistance.

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Zinc in depressive disorder

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Investigations of the cellular immunity during depression and the free interval: evidence for an immune activation in affective psychosis

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Predictive clinical and laboratory parameters for serum zinc and copper in rheumatoid arthritis

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Cytokine production and serum proteins in depression

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Increased CD56+ natural killer cells and related cytokines in major depression

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7 January 2021

Indicators of immune activation in major depression

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7 January 2021

Concentration and microheterogeneity glycophorms of alpha-1-acid glycoprotein in major depressive disorder.

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7 January 2021

Changes in immunoglobulin, complement and acute phase protein levels in the depressed patients and normal controls

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The inflammatory response system and the availability of plasma tryptophan in patients with primary sleep disorders and major depression.

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10.1016/S0165-0327(99)00011-7

Identifiers

DOI

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4 November 2019

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