Intestinal Threonine Utilization for Protein and Mucin Synthesis Is Decreased in Formula-Fed Preterm Pigs (Q56835384)

7

Henk Schierbeek

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Johannes B van Goudoever

12

Johannes B. van Goudoever

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Adrianus C J M de Bruijn

5

Adrianus C. J. M. de Bruijn

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Ingrid B Renes

4

Ingrid B. Renes

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Patrycja J Puiman

1

Patrycja J. Puiman

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Günther Boehm

9

Günther Boehm

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author name string

Mikkel Jensen

2

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Barbara Stoll

3

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Kristien Dorst

6

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Mette Schmidt

8

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Douglas G. Burrin

Necrotising enterocolitis

10

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language of work or name

English

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publication date

18 May 2011

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published in

Journal of Nutrition

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volume

141

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issue

7

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page(s)

1306-1311

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cites work

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Enteral feeding induces diet-dependent mucosal dysfunction, bacterial proliferation, and necrotizing enterocolitis in preterm pigs on parenteral nutrition

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Carbohydrate maldigestion induces necrotizing enterocolitis in preterm pigs

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Diet- and Colonization-Dependent Intestinal Dysfunction Predisposes to Necrotizing Enterocolitis in Preterm Pigs

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Muc2-deficient mice spontaneously develop colitis, indicating that MUC2 is critical for colonic protection

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MUC1 cell surface mucin is a critical element of the mucosal barrier to infection

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The pattern of intestinal substrate oxidation is altered by protein restriction in pigs

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Intestinal inflammation increases gastrointestinal threonine uptake and mucin synthesis in enterally fed minipigs.

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