CDS1 and promoter single nucleotide polymorphisms of the CTLA-4 gene in human myasthenia gravis (Q56904621)

2

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Q Qiu

3

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R Giscombe

4

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DeRen Huang

5

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R Pirskanen

6

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A K Lefvert

https://scigraph.springernature.com/pub.10.1038/sj.gene.6363816

7

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language of work or name

English

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publication date

February 2002

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published in

Genes and Immunity

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volume

3

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issue

1

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page(s)

46-9

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exact match

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cites work

Loss of CTLA-4 leads to massive lymphoproliferation and fatal multiorgan tissue destruction, revealing a critical negative regulatory role of CTLA-4

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7 January 2021

An Mse I RFLP in the human CTLA4 promotor.

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Genetic association of Ctla-4 to myasthenia gravis with thymoma.

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Dinucleotide repeat expansion in the CTLA-4 gene leads to T cell hyper-reactivity via the CD28 pathway in myasthenia gravis

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7 January 2021

CTLA-4 promoter variants in patients with Graves' disease and Hashimoto's thyroiditis.

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7 January 2021

CTLA-4 dimorphisms in gammopathy-associated peripheral neuropathy

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7 January 2021

CTLA4 codon 17 dimorphism in patients with rheumatoid arthritis

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7 January 2021

CTLA4 gene polymorphism correlates with the mode of onset and presence of ICA512 Ab in Japanese type 1 diabetes

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7 January 2021

CTLA-4 gene polymorphism confers susceptibility to primary biliary cirrhosis

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7 January 2021

The CTLA-4 gene is associated with multiple sclerosis.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.GENE.6363816

7 January 2021

The cytotoxic T lymphocyte antigen-4 is a major Graves' disease locus.

1 reference

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7 January 2021

CTLA-4 gene polymorphism at position 49 in exon 1 reduces the inhibitory function of CTLA-4 and contributes to the pathogenesis of Graves' disease

1 reference

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7 January 2021

Determination of acetylcholine receptor antibody in myasthenia gravis: clinical usefulness and pathogenetic implications

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7 January 2021