Endogenous and exogenous pentraxin-3 limits postischemic acute and chronic kidney injury (Q56942220)

Hans-Joachim Anders

13

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Alberto Mantovani

Alberto Mantovani

12

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Cecilia Garlanda

11

Cecilia Garlanda

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Maciej Lech

1

Maciej Lech

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Onkar P Kulkarni

5

Onkar P. Kulkarni

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Heni Eka Susanti

4

Heni Eka Susanti

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Fritz Krombach

10

Fritz Krombach

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author name string

Regina Gröbmayr

3

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Shijun Wang

6

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Hermann-Josef Gröne

7

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Bernd Uhl

8

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Christoph Reichel

9

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language of work or name

English

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publication date

April 2013

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number of pages

15

1 reference

https://scigraph.springernature.com/pub.10.1038/ki.2012.463

inferred from page(s)

published in

Kidney International

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volume

83

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issue

4

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page(s)

647-661

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exact match

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cites work

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Endothelial pentraxin 3 contributes to murine ischemic acute kidney injury

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Biochemical and functional characterization of the interaction between pentraxin 3 and C1q.

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Binding of the long pentraxin PTX3 to factor H: interacting domains and function in the regulation of complement activation

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The tissue pentraxin PTX3 limits C1q-mediated complement activation and phagocytosis of apoptotic cells by dendritic cells

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Endogenous PTX3 translocates at the membrane of late apoptotic human neutrophils and is involved in their engulfment by macrophages

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The prototypic tissue pentraxin PTX3, in contrast to the short pentraxin serum amyloid P, inhibits phagocytosis of late apoptotic neutrophils by macrophages

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Lack of the long pentraxin PTX3 promotes autoimmune lung disease but not glomerulonephritis in murine systemic lupus erythematosus

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Toll-like receptor 4 regulates early endothelial activation during ischemic acute kidney injury

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Chemokines and chemokine receptors are involved in the resolution or progression of renal disease.

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Ischemia reperfusion induces IFN regulatory factor 4 in renal dendritic cells, which suppresses postischemic inflammation and prevents acute renal failure

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A novel iron chelator in combination with a P-selectin antagonist prevents ischemia/reperfusion injury in a rat liver model

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The long pentraxin PTX3 is crucial for tissue inflammation after intestinal ischemia and reperfusion in mice

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Tir8/Sigirr prevents murine lupus by suppressing the immunostimulatory effects of lupus autoantigens.

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Chemokine receptors Ccr1, Ccr2, and Ccr5 mediate neutrophil migration to postischemic tissue

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