Hyperosmotic stress-dependent NFκB activation is regulated by reactive oxygen species and IGF-1 in cultured cardiomyocytes (Q57244730)

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Mario Chiong

object named as

Mario Chiong

7

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author name string

Verónica Eisner

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Alfredo Criollo

2

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Clara Quiroga

3

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Claudio Olea-Azar

4

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Juan Francisco Santibañez

5

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Guillermo Díaz-Araya

8

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Rocío Foncea

The NF-kappa B and I kappa B proteins: new discoveries and insights

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language of work or name

English

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publication date

20 July 2006

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published in

FEBS Letters

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volume

580

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issue

18

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page(s)

4495-4500

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cites work

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An essential role for NF-kappaB in preventing TNF-alpha-induced cell death

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Hyperosmotic stress activates p65/RelB NFkappaB in cultured cardiomyocytes with dichotomic actions on caspase activation and cell death

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Aldose reductase induced by hyperosmotic stress mediates cardiomyocyte apoptosis: differential effects of sorbitol and mannitol

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Redox regulation of transcriptional activators

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Epidermal growth factor activates nuclear factor-kappaB in human proximal tubule cells

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Insulin-like growth factor I as a cardiac hormone: physiological and pathophysiological implications in heart disease

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IGF-I is a matter of heart

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Rapid detection of octamer binding proteins with 'mini-extracts', prepared from a small number of cells

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Inhibition of the nuclear factor kappa B (NF-kappa B) pathway by tetracyclic kaurene diterpenes in macrophages. Specific effects on NF-kappa B-inducing kinase activity and on the coordinate activation of ERK and p38 MAPK.

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Spin trapping of superoxide and hydroxyl radical: practical aspects

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Nox 2 stimulates muscle differentiation via NF-kappaB/iNOS pathway

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Nox2 and Rac1 regulate H2O2-dependent recruitment of TRAF6 to endosomal interleukin-1 receptor complexes

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Dehydration activates an NF-kappaB-driven, COX2-dependent survival mechanism in renal medullary interstitial cells

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Insulin-like Growth Factor-I Rapidly Activates Multiple Signal Transduction Pathways in Cultured Rat Cardiac Myocytes

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PAK2 is cleaved and activated during hyperosmotic shock-induced apoptosis via a caspase-dependent mechanism: Evidence for the involvement of oxidative stress

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Insulin-like growth factor-I protects granule neurons from apoptosis and improves ataxia in weaver mice

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