Population prevalence of APOE, APOC3 and PPAR-? mutations associated to hypertriglyceridemia in French Canadians (Q57316328)

1

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Samuel Aubert

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J�r�me Laroche

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Jo�l Girouard

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Jean Bergeron

6

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Fran�ois Rousseau

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Pierre Julien

Journal of Human Genetics

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publication date

12 November 2004

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published in

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volume

49

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issue

12

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page(s)

691-700

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New genetic variants in the apoA-I and apoC-III genes and familial combined hyperlipidemia

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An apolipoprotein CIII haplotype protective against hypertriglyceridemia is specified by promoter and 3' untranslated region polymorphisms

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Hypertriglyceridemia and the apolipoprotein CIII gene locus: lack of association with the variant insulin response element in Italian school children

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Apolipoprotein E gene polymorphisms and serum cholesterol in healthy Irish adults: a proposed genetic marker for coronary artery disease risk.

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Polymorphisms in the insulin response element of APOC-III gene promoter influence the correlation between insulin and triglycerides or triglyceride-rich lipoproteins in humans

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Inhibitory effects of specific apolipoprotein C-III isoforms on the binding of triglyceride-rich lipoproteins to the lipolysis-stimulated receptor

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Strong association of a single nucleotide substitution in the 3'-untranslated region of the apolipoprotein-CIII gene with common hypertriglyceridemia in Arabs

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LPL polymorphism predicts stroke risk in men

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Common genetic variation in the promoter of the human apo CIII gene abolishes regulation by insulin and may contribute to hypertriglyceridemia

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Apolipoprotein E polymorphism in a Danish population compared to findings in 45 other study populations around the world

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Variation at the apo AI/CIII/AIV gene complex is associated with elevated plasma levels of apo CIII

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Molecular scanning of the human PPARa gene: association of the L162v mutation with hyperapobetalipoproteinemia.

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Mode of action of peroxisome proliferators as hypolipidemic drugs. Suppression of apolipoprotein C-III.

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Multiple Genetic Determinants of Variation of Plasma Lipoproteins in Alberta Hutterites

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Promoter elements and factors required for hepatic and intestinal transcription of the human ApoCIII gene

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Common genomic variation in the APOC3 promoter associated with variation in plasma lipoproteins

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Effect of apolipoprotein E, peroxisome proliferator-activated receptor alpha and lipoprotein lipase gene mutations on the ability of fenofibrate to improve lipid profiles and reach clinical guideline targets among hypertriglyceridemic patients

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DNA POLYMORPHISM ADJACENT TO HUMAN APOPROTEIN A-1 GENE: RELATION TO HYPERTRIGLYCERIDAEMIA

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DNA restriction fragment length polymorphisms as markers of familial coronary heart disease

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An apolipoprotein CIII marker associated with hypertriglyceridemia in Caucasians also confers increased risk in a west Japanese population

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Apolipoprotein E polymorphism in a French Canadian population of northeastern Quebec: allele frequencies and effects on blood lipid and lipoprotein levels.

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Role of ApoCs in lipoprotein metabolism: functional differences between ApoC1, ApoC2, and ApoC3

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Restriction fragment length polymorphisms of the apolipoprotein A-I, C-III, A-IV gene locus Relationships with lipids, apolipoproteins, and premature coronary artery disease

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Plasma levels of remnant particles are determined in part by variation in the APOC3 gene insulin response element and the APOCI-APOE cluster

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Influences of common variants of apolipoprotein E on measures of lipid metabolism in a sample selected for health

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Increased risk for endogenous hypertriglyceridaemia is associated with an apolipoprotein C3 haplotype specified by the SstI polymorphism

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A polymorphism, L162V, in the peroxisome proliferator-activated receptor alpha (PPARalpha) gene is associated with lower body mass index in patients with non-insulin-dependent diabetes mellitus

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Association between the PPARA L162V polymorphism and plasma lipid levels: the Framingham Offspring Study

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Complex genetic contribution of the Apo AI-CIII-AIV gene cluster to familial combined hyperlipidemia. Identification of different susceptibility haplotypes

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