Polymorphism analysis suggests that the gelatinase B gene is not a susceptibility factor for multiple sclerosis (Q57584581)

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4 November 2019

title

Polymorphism analysis suggests that the gelatinase B gene is not a susceptibility factor for multiple sclerosis (English)

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4 November 2019

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2

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4 November 2019

Ghislain Opdenakker

7

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4 November 2019

author name string

I Nelissen

1

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4 November 2019

P Fiten

3

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4 November 2019

J Hillert

4

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4 November 2019

T Olsson

5

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4 November 2019

M G Marrosu

6

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4 November 2019

publication date

1 June 2000

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Journal of Neuroimmunology

4 November 2019

published in

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4 November 2019

volume

105

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4 November 2019

issue

1

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4 November 2019

page(s)

58-63

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Matrix metalloproteinase expression in an experimentally-induced DTH model of multiple sclerosis in the rat CNS.

4 November 2019

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The Expression of Tissue-type Plasminogen Activator, Matrix Metalloproteases and Endogenous Inhibitors in the Central Nervous System in Multiple Sclerosis

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Microsatellite polymorphisms in the gene promoter of monocyte chemotactic protein-3 and analysis of the association between monocyte chemotactic protein-3 alleles and multiple sclerosis development

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Gelatinase in the cerebrospinal fluid of patients with multiple sclerosis and other inflammatory neurological disorders

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Gelatinase B is present in the cerebrospinal fluid during experimental autoimmune encephalomyelitis and cleaves myelin basic protein.

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Suppression of experimental allergic encephalomyelitis in the Lewis rat by the matrix metalloproteinase inhibitor Ro31-9790

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Serum gelatinase B, TIMP-1 and TIMP-2 levels in multiple sclerosis. A longitudinal clinical and MRI study

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Matrix metalloproteinase-9 (gelatinase B) is selectively elevated in CSF during relapses and stable phases of multiple sclerosis

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Effective treatment of models of multiple sclerosis by matrix metalloproteinase inhibitors.

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Reassignment of the 92-kDa type IV collagenase gene (CLG4B) to human chromosome 20

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Matrix metalloproteinases in the normal human central nervous system, microglial nodules, and multiple sclerosis lesions

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Multiple sclerosis in Sardinia is associated and in linkage disequilibrium with HLA-DR3 and -DR4 alleles

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7 January 2021

Leukocyte Gelatinase B Cleavage Releases Encephalitogens from Human Myelin Basic Protein

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7 January 2021

The genetic analysis of multiple sclerosis

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7 January 2021

A genome screen in multiple sclerosis reveals susceptibility loci on chromosome 6p21 and 17q22

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7 January 2021

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Identifiers

DOI

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4 November 2019

1 reference