Plasminogen activator inhibitor-1 deficiency has renal benefits but some adverse systemic consequences in diabetic mice (Q57741361)

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Shannon L Alexander

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Jesus M Lopez-Guisa

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Xiaohe Cai

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Streamson C Chua

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Guoqiang Zhang

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Daryl M Okamura

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Shunya Matsuo

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Allison A Eddy

Nephron Experimental Nephrology

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language of work or name

English

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publication date

2006

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published in

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volume

104

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issue

1

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page(s)

e23-34

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cites work

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PAI-1 deficiency attenuates the fibrogenic response to ureteral obstruction

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Urokinase receptor deficiency accelerates renal fibrosis in obstructive nephropathy.

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Stromal cells are the main plasminogen activator inhibitor-1-producing cells in human fat: evidence of differences between visceral and subcutaneous deposits.

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Plasminogen activator inhibitor-1 is a major stress-regulated gene: implications for stress-induced thrombosis in aged individuals.

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Divergent roles for p55 and p75 TNF-alpha receptors in the induction of plasminogen activator inhibitor-1.

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Hyperglycemia-induced mitochondrial superoxide overproduction activates the hexosamine pathway and induces plasminogen activator inhibitor-1 expression by increasing Sp1 glycosylation

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Oxidative stress activates the plasminogen activator inhibitor type 1 (PAI-1) promoter through an AP-1 response element and cooperates with insulin for additive effects on PAI-1 transcription

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Cellular Carbonyl Stress Enhances the Expression of Plasminogen Activator Inhibitor-1 in Rat White Adipocytes via Reactive Oxygen Species-dependent Pathway

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C-reactive protein increases plasminogen activator inhibitor-1 expression and activity in human aortic endothelial cells: implications for the metabolic syndrome and atherothrombosis.

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Expression of type-1 plasminogen activator inhibitor in the kidney of diabetic rat models

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Plasminogen activator inhibitor-1 deficiency retards diabetic nephropathy

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Evidence that the diabetes gene encodes the leptin receptor: identification of a mutation in the leptin receptor gene in db/db mice

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Tumor necrosis factor alpha is a key component in the obesity-linked elevation of plasminogen activator inhibitor 1

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Amelioration of diabetic nephropathy in SPARC-null mice

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The plasminogen activation system reduces fibrosis in the lung by a hepatocyte growth factor-dependent mechanism

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Suppressions of chronic glomerular injuries and TGF-β1production by HGF in attenuation of murine diabetic nephropathy

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Signaling at the slit diaphragm

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Podocyte migration during nephrotic syndrome requires a coordinated interplay between cathepsin L and alpha3 integrin

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Direct binding of occupied urokinase receptor (uPAR) to LDL receptor-related protein is required for endocytosis of uPAR and regulation of cell surface urokinase activity

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Influence of PAI-1 on adipose tissue growth and metabolic parameters in a murine model of diet-induced obesity.

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