Fecal HMGB1 Is a Novel Marker of Intestinal Mucosal Inflammation in Pediatric Inflammatory Bowel Disease (Q57804107)

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scholarly article by Roberta Vitali et al published 26 July 2011 in The American Journal of Gastroenterology

Language	Label	Description	Also known as
English	Fecal HMGB1 Is a Novel Marker of Intestinal Mucosal Inflammation in Pediatric Inflammatory Bowel Disease	scholarly article by Roberta Vitali et al published 26 July 2011 in The American Journal of Gastroenterology

Statements

scholarly article

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Fecal HMGB1 Is a Novel Marker of Intestinal Mucosal Inflammation in Pediatric Inflammatory Bowel Disease (English)

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based on heuristic

inferred from title

inflammatory bowel diseases

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based on heuristic

inferred from title

object named as

Roberta Vitali

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Laura Stronati

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Giovanni Di Nardo

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Giovanni Di Nardo

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Paolo Rossi

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Salvatore Cucchiara

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Salvatore Cucchiara

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object named as

Anna Negroni

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author name string

Maria Pierdomenico

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Emanuela del Giudice

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language of work or name

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publication date

26 July 2011

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The American Journal of Gastroenterology

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106

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2029-2040

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11

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Endogenous damage-associated molecular pattern molecules at the crossroads of inflammation and cancer

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DAMPs, PAMPs and alarmins: all we need to know about danger

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Hydrophobicity: an ancient damage-associated molecular pattern that initiates innate immune responses.

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HMG-1 as a late mediator of endotoxin lethality in mice

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HMG1 and 2: architectural DNA-binding proteins

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Targeting HMGB1 in inflammation

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High-mobility group box 1 (HMGB1) protein at the crossroads between innate and adaptive immunity

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High-mobility group box 1 (HMGB1) protein: friend and foe.

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High-mobility group box 1 protein (HMGB1): nuclear weapon in the immune arsenal

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The nuclear protein HMGB1 is secreted by monocytes via a non-classical, vesicle-mediated secretory pathway

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HMGB1 as a cytokine and therapeutic target

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Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

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HMGB1 and RAGE in inflammation and cancer

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Extracellular role of HMGB1 in inflammation and sepsis.

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The role of HMGB1 in the pathogenesis of rheumatic disease

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Damage-associated molecular patterns--emerging targets for biologic therapy of childhood arthritides

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High Mobility Group Box 1: a potential therapeutic target for systemic lupus erythematosus.

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Sepsis, ghrelin, the cholinergic anti-inflammatory pathway, gut mucosal hyperpermeability, and high-mobility group box 1.

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Bile high-mobility group box 1 contributes to gut barrier dysfunction in experimental endotoxemia.

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HMGB1 B box increases the permeability of Caco-2 enterocytic monolayers and impairs intestinal barrier function in mice

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Ethyl pyruvate decreases HMGB1 release and ameliorates murine colitis

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Differentiating ulcerative colitis from Crohn disease in children and young adults: report of a working group of the North American Society for Pediatric Gastroenterology, Hepatology, and Nutrition and the Crohn's and Colitis Foundation of America

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Development, validation, and evaluation of a pediatric ulcerative colitis activity index: a prospective multicenter study.

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Comparison of several activity indices for the evaluation of endoscopic activity in UC: inter- and intraobserver consistency.

1 reference

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Post-translational methylation of high mobility group box 1 (HMGB1) causes its cytoplasmic localization in neutrophils.

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Anti-high-mobility group box chromosomal protein 1 antibodies improve survival of rats with sepsis.

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The N-terminal domain of thrombomodulin sequesters high-mobility group-B1 protein, a novel antiinflammatory mechanism

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The HMGB1 receptor RAGE mediates ischemic brain damage

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The nuclear factor HMGB1 mediates hepatic injury after murine liver ischemia-reperfusion

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Protective effect of HMGB1 a box on organ injury of acute pancreatitis in mice

1 reference

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Contributions of high mobility group box protein in experimental and clinical acute lung injury.

1 reference

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Role of endoscopy in predicting the disease course in inflammatory bowel disease

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Monocytic cells hyperacetylate chromatin protein HMGB1 to redirect it towards secretion

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Nucleocytoplasmic shuttling of HMGB1 is regulated by phosphorylation that redirects it toward secretion

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HMGB1 is phosphorylated by classical protein kinase C and is secreted by a calcium-dependent mechanism

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21 January 2018

Identifiers

10.1038/AJG.2011.231

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